Depletion of hypothalamic hypocretin/orexin neurons correlates withimpaired memory in a Parkinson's disease animal model (2020)
- Authors:
- USP affiliated authors: MOREIRA, THIAGO DOS SANTOS - ICB ; MOREIRA, ANA CAROLINA TAKAKURA - ICB
- Unidade: ICB
- DOI: 10.1016/j.expneurol.2019.113110
- Subjects: FISIOLOGIA; FARMACOLOGIA; HIPOTÁLAMO; DOENÇAS NEURODEGENERATIVAS; MEMÓRIA; MODELOS ANIMAIS DE DOENÇAS; DOENÇA DE PARKINSON; RECEPTORES DOPAMINÉRGICOS; HIPOCAMPU DE ANIMAL
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Experimental Neurology
- ISSN: 1090-2430
- Volume/Número/Paginação/Ano: v. 323, art. 113110, 12 p., 2020
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
OLIVEIRA, Luis M. et al. Depletion of hypothalamic hypocretin/orexin neurons correlates withimpaired memory in a Parkinson's disease animal model. Experimental Neurology, v. 323, p. 12 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2019.113110. Acesso em: 30 jul. 2024. -
APA
Oliveira, L. M., Henrique, E., Butelli, I. B., Netto, N. F. C., Caetano, A. L., Moreira, T. dos S., & Takakura, A. C. (2020). Depletion of hypothalamic hypocretin/orexin neurons correlates withimpaired memory in a Parkinson's disease animal model. Experimental Neurology, 323, 12 . doi:10.1016/j.expneurol.2019.113110 -
NLM
Oliveira LM, Henrique E, Butelli IB, Netto NFC, Caetano AL, Moreira T dos S, Takakura AC. Depletion of hypothalamic hypocretin/orexin neurons correlates withimpaired memory in a Parkinson's disease animal model [Internet]. Experimental Neurology. 2020 ; 323 12 .[citado 2024 jul. 30 ] Available from: https://doi.org/10.1016/j.expneurol.2019.113110 -
Vancouver
Oliveira LM, Henrique E, Butelli IB, Netto NFC, Caetano AL, Moreira T dos S, Takakura AC. Depletion of hypothalamic hypocretin/orexin neurons correlates withimpaired memory in a Parkinson's disease animal model [Internet]. Experimental Neurology. 2020 ; 323 12 .[citado 2024 jul. 30 ] Available from: https://doi.org/10.1016/j.expneurol.2019.113110 - P2Y1 receptors expressed by C1 neurons determine peripheral chemoreceptor modulation of breathing, sympathetic activity, and blood pressure
- Acute exercise-induced activation of Phox2b-expressing neurons of the retrotrapezoid nucleus in rats may involve the hypothalamus
- Respiratory and sympathetic chemoreflex regulation by Kölliker-Fuse neurons in rats
- Pontine Kölliker-Fuse neurons are important for chemosensory control in conscious rats
- Activation of central α2-adrenoceptors mediates salivary gland vasoconstriction
- Brainstem areas activated by intermittent apnea in awake unrestrained rats
- α1- and α2-adrenergic receptors in the retrotrapezoid nucleus differentially regulate breathing in anesthetized adult rats
- Inhibition of the pontine Kölliker-Fuse nucleus reduces genioglossal activity elicited by stimulation of the retrotrapezoid chemoreceptor neurons
- Selective lesion of retrotrapezoide Phox2b - Expressing neurons attenuates the central chemoreflex in conscious rats
- Influence of induced obesity on bone mineralization in rats
Informações sobre o DOI: 10.1016/j.expneurol.2019.113110 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
