Human papillomavirus 16 E7 promotes EGFR/PI3K/AKT1/NRF2 signaling pathway contributing to PIR/NF-κB activation in oral cancer cells (2020)
- Authors:
- USP affiliated authors: PIERULIVO, ENRIQUE MARIO BOCCARDO - ICB ; LINO, VANESCA DE SOUZA - ICB
- Unidade: ICB
- DOI: 10.3390/cancers12071904
- Subjects: MICROBIOLOGIA; CÉLULAS CULTIVADAS DE TUMOR; HPV; ONCOPROTEÍNAS; CARCINOMA; ESTRESSE OXIDATIVO; METÁSTASE NEOPLÁSICA; INFECÇÕES POR PAPILLOMAVIRUS
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- Language: Inglês
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- Este artigo é de acesso aberto
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- Licença: cc-by
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ABNT
BELTRÁN, Diego Carrillo et al. Human papillomavirus 16 E7 promotes EGFR/PI3K/AKT1/NRF2 signaling pathway contributing to PIR/NF-κB activation in oral cancer cells. Cancers, v. 12, n. 7, p. 22 , 2020Tradução . . Disponível em: https://doi.org/10.3390/cancers12071904. Acesso em: 04 ago. 2025. -
APA
Beltrán, D. C., Muñoz, J. P., Vásquez, N. G., Blanco, R., León, O., Lino, V. de S., et al. (2020). Human papillomavirus 16 E7 promotes EGFR/PI3K/AKT1/NRF2 signaling pathway contributing to PIR/NF-κB activation in oral cancer cells. Cancers, 12( 7), 22 . doi:10.3390/cancers12071904 -
NLM
Beltrán DC, Muñoz JP, Vásquez NG, Blanco R, León O, Lino V de S, Tapia JC, Maldonado E, Dubois CKD, Hermoso MA, Corvalán AH, Calaf GM, Boccardo E, Aguayo F. Human papillomavirus 16 E7 promotes EGFR/PI3K/AKT1/NRF2 signaling pathway contributing to PIR/NF-κB activation in oral cancer cells [Internet]. Cancers. 2020 ; 12( 7): 22 .[citado 2025 ago. 04 ] Available from: https://doi.org/10.3390/cancers12071904 -
Vancouver
Beltrán DC, Muñoz JP, Vásquez NG, Blanco R, León O, Lino V de S, Tapia JC, Maldonado E, Dubois CKD, Hermoso MA, Corvalán AH, Calaf GM, Boccardo E, Aguayo F. Human papillomavirus 16 E7 promotes EGFR/PI3K/AKT1/NRF2 signaling pathway contributing to PIR/NF-κB activation in oral cancer cells [Internet]. Cancers. 2020 ; 12( 7): 22 .[citado 2025 ago. 04 ] Available from: https://doi.org/10.3390/cancers12071904 - Interplay between Epstein-Barr virus infection and environmental xenobiotic exposure in cancer
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Informações sobre o DOI: 10.3390/cancers12071904 (Fonte: oaDOI API)
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