Genetic association between HLA-G 14-bp polymorphism and diseases: a systematic review and meta-analysis (2018)
- Authors:
- USP affiliated authors: MENDES JUNIOR, CELSO TEIXEIRA - FFCLRP ; DONADI, EDUARDO ANTONIO - FMRP ; ALMEIDA, BIBIANA SGORLA DE - FMRP
- Unidades: FFCLRP; FMRP
- DOI: 10.1016/j.humimm.2018.08.003
- Subjects: POLIMORFISMO; ALELOS; LÚPUS ERITEMATOSO SISTÊMICO; MUTAÇÃO
- Keywords: Non-classical MHC; HLA-G; 14-bp polymorphism; Indel; 3′UTR
- Agências de fomento:
- Language: Inglês
- Source:
- Título: Human Immunology
- ISSN: 0198-8859
- Volume/Número/Paginação/Ano: v. 79, n. 10, p. 724-735, 2018
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
ALMEIDA, Bibiana Sgorla de et al. Genetic association between HLA-G 14-bp polymorphism and diseases: a systematic review and meta-analysis. Human Immunology, v. 79, n. 10, p. 724-735, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.humimm.2018.08.003. Acesso em: 02 abr. 2026. -
APA
Almeida, B. S. de, Muniz, Y. C. N., Prompt, A. H., Castelli, E. C., Mendes Júnior, C. T., & Donadi, E. A. (2018). Genetic association between HLA-G 14-bp polymorphism and diseases: a systematic review and meta-analysis. Human Immunology, 79( 10), 724-735. doi:10.1016/j.humimm.2018.08.003 -
NLM
Almeida BS de, Muniz YCN, Prompt AH, Castelli EC, Mendes Júnior CT, Donadi EA. Genetic association between HLA-G 14-bp polymorphism and diseases: a systematic review and meta-analysis [Internet]. Human Immunology. 2018 ; 79( 10): 724-735.[citado 2026 abr. 02 ] Available from: https://doi.org/10.1016/j.humimm.2018.08.003 -
Vancouver
Almeida BS de, Muniz YCN, Prompt AH, Castelli EC, Mendes Júnior CT, Donadi EA. Genetic association between HLA-G 14-bp polymorphism and diseases: a systematic review and meta-analysis [Internet]. Human Immunology. 2018 ; 79( 10): 724-735.[citado 2026 abr. 02 ] Available from: https://doi.org/10.1016/j.humimm.2018.08.003 - Haplotypes of the HLA-G 3’ untranslated region respond to endogenous factors of HLA-G+ and HLA-G- cell lines differentially
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