Enhancement of endocannabinoid-dependent depolarization-induced suppression of excitation in glycinergic neurons by prolonged exposure to high doses of salicylate (2018)
- Authors:
- Autor USP: LEÃO, RICARDO MAURICIO XAVIER - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.neuroscience.2018.02.016
- Subjects: CÓCLEA; SINAPSE; CANABINOIDES
- Keywords: DSE; Cochlear nucleus; Endocannabinoid; Salicylate; Synapse; Tinnitus
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Neuroscience
- ISSN: 0306-4522
- Volume/Número/Paginação/Ano: v. 376, p. 72-79, 2018
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
ZUGAIB, João e LEÃO, Ricardo Maurício Xavier. Enhancement of endocannabinoid-dependent depolarization-induced suppression of excitation in glycinergic neurons by prolonged exposure to high doses of salicylate. Neuroscience, v. 376, p. 72-79, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2018.02.016. Acesso em: 21 jan. 2026. -
APA
Zugaib, J., & Leão, R. M. X. (2018). Enhancement of endocannabinoid-dependent depolarization-induced suppression of excitation in glycinergic neurons by prolonged exposure to high doses of salicylate. Neuroscience, 376, 72-79. doi:10.1016/j.neuroscience.2018.02.016 -
NLM
Zugaib J, Leão RMX. Enhancement of endocannabinoid-dependent depolarization-induced suppression of excitation in glycinergic neurons by prolonged exposure to high doses of salicylate [Internet]. Neuroscience. 2018 ; 376 72-79.[citado 2026 jan. 21 ] Available from: https://doi.org/10.1016/j.neuroscience.2018.02.016 -
Vancouver
Zugaib J, Leão RMX. Enhancement of endocannabinoid-dependent depolarization-induced suppression of excitation in glycinergic neurons by prolonged exposure to high doses of salicylate [Internet]. Neuroscience. 2018 ; 376 72-79.[citado 2026 jan. 21 ] Available from: https://doi.org/10.1016/j.neuroscience.2018.02.016 - Electrophysiological and molecular properties of neuronal populations in an auditory nucleu of songbirds
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Informações sobre o DOI: 10.1016/j.neuroscience.2018.02.016 (Fonte: oaDOI API)
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