Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection (2018)
- Authors:
- USP affiliated authors: NASCIMENTO, ROGERIO SILVA DO - ICB ; LIMA, MARIA REGINA D'IMPERIO - ICB ; MOSIG, JOSE MARIA ALVAREZ - ICB
- Unidade: ICB
- DOI: 10.3389/fimmu.2018.00997
- Subjects: DOENÇA DE CHAGAS EM ANIMAL; TRYPANOSOMA CRUZI; CÉLULAS MORTAS; CAMUNDONGOS; LEUCOCITOSE ANIMAL; IMUNOSSUPRESSÃO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 9, p. 1-10, 2018
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
FONSECA, Raissa et al. Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection. Frontiers in Immunology, v. 9, p. 1-10, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00997. Acesso em: 13 fev. 2026. -
APA
Fonseca, R., Salgado, R. M., Silva, H. B., Nascimento, R. S. do, Lima, M. R. D. 'I., & Alvarez, J. M. (2018). Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection. Frontiers in Immunology, 9, 1-10. doi:10.3389/fimmu.2018.00997 -
NLM
Fonseca R, Salgado RM, Silva HB, Nascimento RS do, Lima MRD'I, Alvarez JM. Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection [Internet]. Frontiers in Immunology. 2018 ; 9 1-10.[citado 2026 fev. 13 ] Available from: https://doi.org/10.3389/fimmu.2018.00997 -
Vancouver
Fonseca R, Salgado RM, Silva HB, Nascimento RS do, Lima MRD'I, Alvarez JM. Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection [Internet]. Frontiers in Immunology. 2018 ; 9 1-10.[citado 2026 fev. 13 ] Available from: https://doi.org/10.3389/fimmu.2018.00997 - Cellular renewal and improvement of local cell effector activity in peritoneal cavity in response to infectious stimuli
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- Characterization of a 250kd molecule of plasmodium chabaudi chabaudi schizont forms present in the serum of acutely infected mice
- Influence of the polyclonal activation induced by plasmodium chabaudi on ongoing OVA-specific B- and T-cell responses
- Role of CD28 in polyclonal and specific T and B cell responses required for protection against blood stage malaria
- Characterization of memory CD4+ T and B cells in the spleen of mice infected with blood stages of Plasmodium chabaundi AS
- IL-2 secretion and CD4+CD25 T cells in polyclonal lymphocyte activation (PLA) induced by Plasmodium chabaudi infection
- IFN-'gama', but not nitric oxide or specific IgG, is essential for the in vivo control of low-virulence Sylvio X10/4 Trypanosoma cruzi parasites
Informações sobre o DOI: 10.3389/fimmu.2018.00997 (Fonte: oaDOI API)
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