Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection (2018)
- Authors:
- USP affiliated authors: NASCIMENTO, ROGERIO SILVA DO - ICB ; LIMA, MARIA REGINA D'IMPERIO - ICB ; MOSIG, JOSE MARIA ALVAREZ - ICB
- Unidade: ICB
- DOI: 10.3389/fimmu.2018.00997
- Subjects: DOENÇA DE CHAGAS EM ANIMAL; TRYPANOSOMA CRUZI; CÉLULAS MORTAS; CAMUNDONGOS; LEUCOCITOSE ANIMAL; IMUNOSSUPRESSÃO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 9, p. 1-10, 2018
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
FONSECA, Raissa et al. Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection. Frontiers in Immunology, v. 9, p. 1-10, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00997. Acesso em: 31 mar. 2026. -
APA
Fonseca, R., Salgado, R. M., Silva, H. B., Nascimento, R. S. do, Lima, M. R. D. 'I., & Alvarez, J. M. (2018). Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection. Frontiers in Immunology, 9, 1-10. doi:10.3389/fimmu.2018.00997 -
NLM
Fonseca R, Salgado RM, Silva HB, Nascimento RS do, Lima MRD'I, Alvarez JM. Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection [Internet]. Frontiers in Immunology. 2018 ; 9 1-10.[citado 2026 mar. 31 ] Available from: https://doi.org/10.3389/fimmu.2018.00997 -
Vancouver
Fonseca R, Salgado RM, Silva HB, Nascimento RS do, Lima MRD'I, Alvarez JM. Programmed cell death protein 1–PDl1 interaction prevents heart damage in chronic Trypanosoma cruzi infection [Internet]. Frontiers in Immunology. 2018 ; 9 1-10.[citado 2026 mar. 31 ] Available from: https://doi.org/10.3389/fimmu.2018.00997 - Cellular renewal and improvement of local cell effector activity in peritoneal cavity in response to infectious stimuli
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