Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure (2017)
- Authors:
- USP affiliated authors: SILVA, CARLOS ALBERTO AGUIAR DA - FMRP ; KETTELHUT, ISIS DO CARMO - FMRP ; RAMOS, SIMONE GUSMÃO - FMRP ; BARUFFI, MARCELO DIAS - FCFRP ; SALGADO, HELIO CESAR - FMRP ; ZINNI, CIBELE MARIA PRADO - FZEA
- Unidades: FMRP; FCFRP; FZEA
- DOI: 10.1371/journal.pone.0189469
- Subjects: BIOMARCADORES; PRESSÃO SANGUÍNEA; CARDIOMEGALIA; DIAGNÓSTICO POR IMAGEM; ECOCARDIOGRAFIA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2017
- Source:
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
PRADO, Fernanda Peres et al. Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure. PLOS ONE, v. 12, n. 12, 2017Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0189469. Acesso em: 25 fev. 2026. -
APA
Prado, F. P., Santos, D. O. dos, Blefari, V., Silva, C. A. A. da, Machado, J., Kettelhut, I. do C., et al. (2017). Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure. PLOS ONE, 12( 12). doi:10.1371/journal.pone.0189469 -
NLM
Prado FP, Santos DO dos, Blefari V, Silva CAA da, Machado J, Kettelhut I do C, Ramos SG, Baruffi MD, Salgado HC, Prado CM. Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure [Internet]. PLOS ONE. 2017 ; 12( 12):[citado 2026 fev. 25 ] Available from: https://doi.org/10.1371/journal.pone.0189469 -
Vancouver
Prado FP, Santos DO dos, Blefari V, Silva CAA da, Machado J, Kettelhut I do C, Ramos SG, Baruffi MD, Salgado HC, Prado CM. Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure [Internet]. PLOS ONE. 2017 ; 12( 12):[citado 2026 fev. 25 ] Available from: https://doi.org/10.1371/journal.pone.0189469 - Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats
- Alterations in adherens junction and gap junction precede desmosomes remodeling during the transition from experimental compensated cardiac hypertrophy to decompensation
- Alterations in adherens junction and Gap junction precede desmosomes remodeling during the transition from experimental compensated cardiac hypertrophy to decompensation
- Atividade do nervo simpático renal na hipertensão induzida por L-NAME, em ratos acordados
- Acetylcholinesterase inhibition attenuates the development of hypertension and inflammation in spontaneously hypertensive rats
- Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure
- Mmp-2/timp-2 imbalance and increased collagen production are crucial to the transition from compensated cardiac hypertrophy to heart failure in rats subjected to abdominal aorta constriction
- Pyridostigmine enhances vagal influence to the heart but increases renal sympathetic nerve activity in anesthetized rats
- Effect of acetylcholinesterase blockade with pyridostigmine on baroreflex and cardiovascular autonomic control in heart failure rats, six to seven weeks after coronary artery ligation
- Analysis of heart rate variability by multiscale entropy in spontaneuously hypertensive rats
Informações sobre o DOI: 10.1371/journal.pone.0189469 (Fonte: oaDOI API)
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