Giant renal angiomyolipoma following ovarian stimulation therapy (2018)
- Authors:
- Autor USP: ONUCHIC, LUIZ FERNANDO - FM
- Unidade: FM
- DOI: 10.1016/j.urology.2017.10.039
- Subjects: NEFROPATIAS; COMPLICAÇÕES NA GRAVIDEZ; ESTRÓGENOS; TOMOGRAFIA COMPUTADORIZADA DE EMISSÃO
- Language: Inglês
- Imprenta:
- Source:
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
WATANABE, Elieser H. et al. Giant renal angiomyolipoma following ovarian stimulation therapy. Urology, v. 112, p. E3-E4, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.urology.2017.10.039. Acesso em: 09 jan. 2026. -
APA
Watanabe, E. H., Neves, P. D., Balbo, B. E., Sampaio, C. A., & Onuchic, L. F. (2018). Giant renal angiomyolipoma following ovarian stimulation therapy. Urology, 112, E3-E4. doi:10.1016/j.urology.2017.10.039 -
NLM
Watanabe EH, Neves PD, Balbo BE, Sampaio CA, Onuchic LF. Giant renal angiomyolipoma following ovarian stimulation therapy [Internet]. Urology. 2018 ; 112 E3-E4.[citado 2026 jan. 09 ] Available from: https://doi.org/10.1016/j.urology.2017.10.039 -
Vancouver
Watanabe EH, Neves PD, Balbo BE, Sampaio CA, Onuchic LF. Giant renal angiomyolipoma following ovarian stimulation therapy [Internet]. Urology. 2018 ; 112 E3-E4.[citado 2026 jan. 09 ] Available from: https://doi.org/10.1016/j.urology.2017.10.039 - Expression of polyductin, the PKHD1 gene product, during normal and abnormal fetal development of the intrahepatic billiary sytem, billiary atresia, paucity of the intrahepatic biliary system and liver tumors
- Polyductin undergoes notch-like processing and regulated release from primary cilia
- Molecular and cellular pathogenesis of autosomal recessive polycystic kidney disease
- Haploinsufficiency for PKD1 results in increased tubularization of the bowman´s capsule and overexpression of COX-2
- Development of in vitro espression system of autosomal ressive polycystic kidney disease responsible gene (PKIID1) product, polydutin
- Inactivation of PKHD1 results in a liver phenotype characteristic of autosomal recessive polycystic kidney disease (ARPKD)
- Dendrogram Analyses of Relative Expression Between Bladder Cancers and Controls Identify PTEN-HIF1α Association as an Alteration Characteristic of Malignancy
- Infundibular stenosis in Bardet–Biedl syndrome
- Clinical aspects of autosomal recessive polycystic kidney disease
- Intragenic motifs regulate the transcriptional complexity of Pkhd1/PKHD1
Informações sobre o DOI: 10.1016/j.urology.2017.10.039 (Fonte: oaDOI API)
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