Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects (2018)
- Authors:
- USP affiliated authors: GRINBERG, LEA TENENHOLZ - FM ; SUEMOTO, CLAUDIA KIMIE - FM ; PINTO, NADJA CRISTHINA DE SOUZA - IQ
- Unidades: FM; IQ
- DOI: 10.1016/j.neurobiolaging.2018.09.015
- Subjects: DNA MITOCONDRIAL; MUTAGÊNESE
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Neurobiology of Aging
- ISSN: 0197-4580
- Volume/Número/Paginação/Ano: v. 73, p. 161-170, 2018
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
SOLTYS, Daniela T; PEREIRA, Carolina P. M; ROWIES, Fernanda T; et al. Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects. Neurobiology of Aging, New York, v. 73, p. 161-170, 2018. Disponível em: < http://dx.doi.org/10.1016/j.neurobiolaging.2018.09.015 > DOI: 10.1016/j.neurobiolaging.2018.09.015. -
APA
Soltys, D. T., Pereira, C. P. M., Rowies, F. T., Rowies, F. T., Grinberg, L. T., Suemoto, C. K., et al. (2018). Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects. Neurobiology of Aging, 73, 161-170. doi:10.1016/j.neurobiolaging.2018.09.015 -
NLM
Soltys DT, Pereira CPM, Rowies FT, Rowies FT, Grinberg LT, Suemoto CK, Leite REP, Rodriguez RD, Ericson NG, Bielas JH, Souza-Pinto NC de. Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects [Internet]. Neurobiology of Aging. 2018 ; 73 161-170.Available from: http://dx.doi.org/10.1016/j.neurobiolaging.2018.09.015 -
Vancouver
Soltys DT, Pereira CPM, Rowies FT, Rowies FT, Grinberg LT, Suemoto CK, Leite REP, Rodriguez RD, Ericson NG, Bielas JH, Souza-Pinto NC de. Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects [Internet]. Neurobiology of Aging. 2018 ; 73 161-170.Available from: http://dx.doi.org/10.1016/j.neurobiolaging.2018.09.015 - Selective Vulnerability of Brainstem Nuclei in Distinct Tauopathies: A Postmortem Study
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- Removal of oxidative DNA damage via FEN1-dependent long-patch base excision repair in human cell mitochondria
- Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane
Informações sobre o DOI: 10.1016/j.neurobiolaging.2018.09.015 (Fonte: oaDOI API)
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