Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects (2018)
- Authors:
- USP affiliated authors: GRINBERG, LEA TENENHOLZ - FM ; SUEMOTO, CLAUDIA KIMIE - FM ; PINTO, NADJA CRISTHINA DE SOUZA - IQ
- Schools: FM; IQ
- DOI: 10.1016/j.neurobiolaging.2018.09.015
- Subjects: DNA MITOCONDRIAL; MUTAGÊNESE
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Neurobiology of Aging
- ISSN: 0197-4580
- Volume/Número/Paginação/Ano: v. 73, p. 161-170, 2018
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
SOLTYS, Daniela T et al. Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects. Neurobiology of Aging, v. 73, p. 161-170, 2018Tradução . . Disponível em: http://dx.doi.org/10.1016/j.neurobiolaging.2018.09.015. Acesso em: 04 jul. 2022. -
APA
Soltys, D. T., Pereira, C. P. M., Rowies, F. T., Rowies, F. T., Grinberg, L. T., Suemoto, C. K., et al. (2018). Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects. Neurobiology of Aging, 73, 161-170. doi:10.1016/j.neurobiolaging.2018.09.015 -
NLM
Soltys DT, Pereira CPM, Rowies FT, Rowies FT, Grinberg LT, Suemoto CK, Leite REP, Rodriguez RD, Ericson NG, Bielas JH, Souza-Pinto NC de. Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects [Internet]. Neurobiology of Aging. 2018 ; 73 161-170.[citado 2022 jul. 04 ] Available from: http://dx.doi.org/10.1016/j.neurobiolaging.2018.09.015 -
Vancouver
Soltys DT, Pereira CPM, Rowies FT, Rowies FT, Grinberg LT, Suemoto CK, Leite REP, Rodriguez RD, Ericson NG, Bielas JH, Souza-Pinto NC de. Lower mitochondrial DNA content but not increased mutagenesis associates with decreased base excision repair activity in brains of AD subjects [Internet]. Neurobiology of Aging. 2018 ; 73 161-170.[citado 2022 jul. 04 ] Available from: http://dx.doi.org/10.1016/j.neurobiolaging.2018.09.015 - Adenovirus-Mediated Transduction of Insulin-Like Growth Factor 1 Protects Hippocampal Neurons from the Toxicity of A beta Oligomers and Prevents Memory Loss in an Alzheimer Mouse Model
- Selective Vulnerability of Brainstem Nuclei in Distinct Tauopathies: A Postmortem Study
- A microdeletion in Alzheimer’s disease disrupts NAMPT gene
- Increased DNA Copy Number Variation Mosaicism in Elderly Human Brain
- Association of rare copy number variations and risk for Alzheimer's disease
- Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane
- The role of mitochondrial DNA damage in the citotoxicity of reactive oxygen species
- Base excision repair activities differ in human lung cancer cells and corresponding normal controls
- ExoMeg1: a new exonuclease from metagenomic library
- Formation and repair of oxidative damage in the mitochondrial DNA
Informações sobre o DOI: 10.1016/j.neurobiolaging.2018.09.015 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas