Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection (2017)
- Authors:
- Autor USP: ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- DOI: 10.1074/jbc.m117.780981
- Subjects: LEISHMANIA; MACRÓFAGOS
- Keywords: AUTOPHAGY; INFECTION; LEISHMANIA; MACROPHAGE; TOLL-LIKE RECEPTOR (TLR)
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Biological Chemistry
- ISSN: 0021-9258
- Volume/Número/Paginação/Ano: v. 292, n. 32, p. 13087-13096, 2017
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
FRANCO, Luís Henrique et al. Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection. Journal of Biological Chemistry, v. 292, n. 32, p. 13087-13096, 2017Tradução . . Disponível em: https://doi.org/10.1074/jbc.m117.780981. Acesso em: 21 fev. 2026. -
APA
Franco, L. H., Fleuri, A. K. A., Pellison, N. C., Quirino, G. F. da S., Horta, C. V., Carvalho, R. V. H. de, et al. (2017). Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection. Journal of Biological Chemistry, 292( 32), 13087-13096. doi:10.1074/jbc.m117.780981 -
NLM
Franco LH, Fleuri AKA, Pellison NC, Quirino GF da S, Horta CV, Carvalho RVH de, Oliveira SC, Zamboni DS. Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection [Internet]. Journal of Biological Chemistry. 2017 ; 292( 32): 13087-13096.[citado 2026 fev. 21 ] Available from: https://doi.org/10.1074/jbc.m117.780981 -
Vancouver
Franco LH, Fleuri AKA, Pellison NC, Quirino GF da S, Horta CV, Carvalho RVH de, Oliveira SC, Zamboni DS. Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection [Internet]. Journal of Biological Chemistry. 2017 ; 292( 32): 13087-13096.[citado 2026 fev. 21 ] Available from: https://doi.org/10.1074/jbc.m117.780981 - Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection
- The role of nod-like receptors and the inflammasome in recognition of the infection by the intracellular bacteria Coxiella burnetii
- Inflammasome mediated innate immune responses to Leishmania
- Inhibition of non-canonical inflammasome activation in response to Legionella pneumophila by a Dot/Icm effector of Coxiella burnetii
- AIM2 engages active but unprocessed caspase-1 to induce noncanonical activation of the NLRP3 inflammasome
- Inflammasome biology taught by Legionella pneumophila
- The NLRP3 inflammasome contributes to host protection during Sporothrix schenckii infection
- O papel de inflamassomas no controle da infecção por diferentes espécies do gênero Legionella
- Participação dos receptores de reconhecimento padrão NOD1 e NOD2 no recrutamento de neutrófilos para o pulmão em resposta a infecção murina por legionella penumophila
- Murine alveolar macrophages are highly susceptible to replication of Coxiella burnetii phase II in vitro
Informações sobre o DOI: 10.1074/jbc.m117.780981 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas