Polymorphisms in SIGLEC1 contribute to susceptibility to pulmonary active tuberculosis possibly through the modulation of IL-1ß (2017)
- Authors:
- USP affiliated authors: PONTILLO, ALESSANDRA - ICB ; BARGIERI, BRUNA CUNHA DE ALENCAR - ICB
- Unidade: ICB
- DOI: 10.1016/j.meegid.2017.09.031
- Subjects: IMUNOLOGIA; POLIMORFISMO; TUBERCULOSE PULMONAR; MACRÓFAGOS; INTERAÇÃO CELULAR
- Language: Inglês
- Imprenta:
- Source:
- Título: Infection, Genetics and Evolution
- ISSN: 1567-7257
- Volume/Número/Paginação/Ano: v. 55, n. 23, p. 313-317, 2017
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
LIMA, Dhemerson Souza de et al. Polymorphisms in SIGLEC1 contribute to susceptibility to pulmonary active tuberculosis possibly through the modulation of IL-1ß. Infection, Genetics and Evolution, v. 55, n. 23, p. 313-317, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.meegid.2017.09.031. Acesso em: 30 dez. 2025. -
APA
Lima, D. S. de, Leal, V. N. C., Ogusku, M. M., Sadahiro, A., Pontillo, A., & Bargieri, B. C. de A. (2017). Polymorphisms in SIGLEC1 contribute to susceptibility to pulmonary active tuberculosis possibly through the modulation of IL-1ß. Infection, Genetics and Evolution, 55( 23), 313-317. doi:10.1016/j.meegid.2017.09.031 -
NLM
Lima DS de, Leal VNC, Ogusku MM, Sadahiro A, Pontillo A, Bargieri BC de A. Polymorphisms in SIGLEC1 contribute to susceptibility to pulmonary active tuberculosis possibly through the modulation of IL-1ß [Internet]. Infection, Genetics and Evolution. 2017 ; 55( 23): 313-317.[citado 2025 dez. 30 ] Available from: https://doi.org/10.1016/j.meegid.2017.09.031 -
Vancouver
Lima DS de, Leal VNC, Ogusku MM, Sadahiro A, Pontillo A, Bargieri BC de A. Polymorphisms in SIGLEC1 contribute to susceptibility to pulmonary active tuberculosis possibly through the modulation of IL-1ß [Internet]. Infection, Genetics and Evolution. 2017 ; 55( 23): 313-317.[citado 2025 dez. 30 ] Available from: https://doi.org/10.1016/j.meegid.2017.09.031 - Sensing of HIV-1 entry triggers a type I interferon response in human primary macrophages
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Informações sobre o DOI: 10.1016/j.meegid.2017.09.031 (Fonte: oaDOI API)
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