Kinin-B1 receptor stimulation promotes invasion and is involved in cell-cell interaction of co-cultured glioblastoma and mesenchymal stem cells (2018)
- Authors:
- Autor USP: ULRICH, ALEXANDER HENNING - IQ
- Unidade: IQ
- DOI: 10.1038/s41598-018-19359-1
- Subjects: GLIOMA; BRADICININA; INFLAMAÇÃO; CÉLULAS-TRONCO
- Language: Inglês
- Imprenta:
- Source:
- Título: Scientific Reports
- ISSN: 2045-2322
- Volume/Número/Paginação/Ano: v. 8, n. 1, p. 1-16 art. 1299, 2018
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
OLIVEIRA, Mona das Neves et al. Kinin-B1 receptor stimulation promotes invasion and is involved in cell-cell interaction of co-cultured glioblastoma and mesenchymal stem cells. Scientific Reports, v. 8, n. 1, p. 1-16 art. 1299, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-19359-1. Acesso em: 19 nov. 2024. -
APA
Oliveira, M. das N., Pillat, M. M., Motaln, H., Ulrich, H., & Lah, T. T. (2018). Kinin-B1 receptor stimulation promotes invasion and is involved in cell-cell interaction of co-cultured glioblastoma and mesenchymal stem cells. Scientific Reports, 8( 1), 1-16 art. 1299. doi:10.1038/s41598-018-19359-1 -
NLM
Oliveira M das N, Pillat MM, Motaln H, Ulrich H, Lah TT. Kinin-B1 receptor stimulation promotes invasion and is involved in cell-cell interaction of co-cultured glioblastoma and mesenchymal stem cells [Internet]. Scientific Reports. 2018 ; 8( 1): 1-16 art. 1299.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1038/s41598-018-19359-1 -
Vancouver
Oliveira M das N, Pillat MM, Motaln H, Ulrich H, Lah TT. Kinin-B1 receptor stimulation promotes invasion and is involved in cell-cell interaction of co-cultured glioblastoma and mesenchymal stem cells [Internet]. Scientific Reports. 2018 ; 8( 1): 1-16 art. 1299.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1038/s41598-018-19359-1 - Aluminum-induced alterations of purinergic signalling in embryonic neural progenitor cells
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Informações sobre o DOI: 10.1038/s41598-018-19359-1 (Fonte: oaDOI API)
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