Topological characterization of the mitochondrial phospholipid scramblase 3 (2017)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1002/1873-3468.12917
- Subjects: MITOCÔNDRIAS; FOSFOLIPÍDEOS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Febs Letters
- ISSN: 0014-5793
- Volume/Número/Paginação/Ano: v. 591, p. 4056-4066, 2017
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
MARTINEZ, Luis Alberto Luevano e KOWALTOWSKI, Alicia Juliana. Topological characterization of the mitochondrial phospholipid scramblase 3. Febs Letters, v. 591, p. 4056-4066, 2017Tradução . . Disponível em: https://doi.org/10.1002/1873-3468.12917. Acesso em: 26 abr. 2024. -
APA
Martinez, L. A. L., & Kowaltowski, A. J. (2017). Topological characterization of the mitochondrial phospholipid scramblase 3. Febs Letters, 591, 4056-4066. doi:10.1002/1873-3468.12917 -
NLM
Martinez LAL, Kowaltowski AJ. Topological characterization of the mitochondrial phospholipid scramblase 3 [Internet]. Febs Letters. 2017 ; 591 4056-4066.[citado 2024 abr. 26 ] Available from: https://doi.org/10.1002/1873-3468.12917 -
Vancouver
Martinez LAL, Kowaltowski AJ. Topological characterization of the mitochondrial phospholipid scramblase 3 [Internet]. Febs Letters. 2017 ; 591 4056-4066.[citado 2024 abr. 26 ] Available from: https://doi.org/10.1002/1873-3468.12917 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1002/1873-3468.12917 (Fonte: oaDOI API)
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