Rewiring metabolism by targeting dicer in adipocytes (2016)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Subjects: METABOLISMO; DIABETES MELLITUS NÃO INSULINO-DEPENDENTE; DIETÉTICA
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
- Publisher place: Natal
- Date published: 2016
- Source:
- Título: Abstracts
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology (SBBq)
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ABNT
REIS, F. C et al. Rewiring metabolism by targeting dicer in adipocytes. 2016, Anais.. Natal: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2016. . Acesso em: 10 out. 2024. -
APA
Reis, F. C., Branquinho, j. l, Brandão, B. B., Guerra, B. A., Amigo, I., Lauritzen, H. P., et al. (2016). Rewiring metabolism by targeting dicer in adipocytes. In Abstracts. Natal: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). -
NLM
Reis FC, Branquinho j. l, Brandão BB, Guerra BA, Amigo I, Lauritzen HP, Thomou T, Silva ID, Frontini A, Cinti S, Kahn CR, Festuccia WTL, Kowaltowski AJ, Mori MA da S. Rewiring metabolism by targeting dicer in adipocytes. Abstracts. 2016 ;[citado 2024 out. 10 ] -
Vancouver
Reis FC, Branquinho j. l, Brandão BB, Guerra BA, Amigo I, Lauritzen HP, Thomou T, Silva ID, Frontini A, Cinti S, Kahn CR, Festuccia WTL, Kowaltowski AJ, Mori MA da S. Rewiring metabolism by targeting dicer in adipocytes. Abstracts. 2016 ;[citado 2024 out. 10 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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