The RelA/cRel nuclear factor-κB (NF-κB) dimer, crucial for inflammation resolution, mediates the transcription of the key enzyme in melatonin synthesis in RAW 264.7 macrophages (2016)
- Authors:
- USP affiliated authors: MUXEL, SANDRA MARCIA - IB ; WINTER, LUCILE MARIA FLOETER - IB ; MARKUS, REGINA PEKELMANN - IB
- Unidade: IB
- DOI: 10.1111/jpi.12321
- Subjects: MELATONINA; MACRÓFAGOS; LEISHMANIA; BIOLOGIA MOLECULAR; PARASITOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Pineal Research
- ISSN: 1600-079X
- Volume/Número/Paginação/Ano: p. on-line, 2016
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
MUXEL, Sandra Marcia et al. The RelA/cRel nuclear factor-κB (NF-κB) dimer, crucial for inflammation resolution, mediates the transcription of the key enzyme in melatonin synthesis in RAW 264.7 macrophages. Journal of Pineal Research, p. on-line, 2016Tradução . . Disponível em: https://doi.org/10.1111/jpi.12321. Acesso em: 07 maio 2025. -
APA
Muxel, S. M., Laranjeira-Silva, M. F., Carvalho-Sousa, C. E., Floeter-Winter, L. M., & Markus, R. P. (2016). The RelA/cRel nuclear factor-κB (NF-κB) dimer, crucial for inflammation resolution, mediates the transcription of the key enzyme in melatonin synthesis in RAW 264.7 macrophages. Journal of Pineal Research, on-line. doi:10.1111/jpi.12321 -
NLM
Muxel SM, Laranjeira-Silva MF, Carvalho-Sousa CE, Floeter-Winter LM, Markus RP. The RelA/cRel nuclear factor-κB (NF-κB) dimer, crucial for inflammation resolution, mediates the transcription of the key enzyme in melatonin synthesis in RAW 264.7 macrophages [Internet]. Journal of Pineal Research. 2016 ; on-line.[citado 2025 maio 07 ] Available from: https://doi.org/10.1111/jpi.12321 -
Vancouver
Muxel SM, Laranjeira-Silva MF, Carvalho-Sousa CE, Floeter-Winter LM, Markus RP. The RelA/cRel nuclear factor-κB (NF-κB) dimer, crucial for inflammation resolution, mediates the transcription of the key enzyme in melatonin synthesis in RAW 264.7 macrophages [Internet]. Journal of Pineal Research. 2016 ; on-line.[citado 2025 maio 07 ] Available from: https://doi.org/10.1111/jpi.12321 - Melatonin attenuates Leishmania (L.) amazonensis infection bymodulating arginine metabolism
- Axenic Leishmania amazonensis promastigotes sense both the external and internal arginine pool distinctly regulating the two transporter-coding genes
- NF-kB drives the synthesis of melatonin in RAW 264.7 macrophages by inducing the transcription of the arylalkylamine-N-acetyltransferase (AA-NAT) gene
- Pinealoctomy associated decrease in ribosomal gene activity in rats
- Arginine and Polyamines Fate in Leishmania Infection
- Transbilayer dynamics of phospholipids in the plasma membrane of the Leishmania genus
- Leishmania amazonensis Arginase compartmentalization in the glycosome is important for parasite infectivity
- Toll-Like Receptor and miRNA-let-7e Expression Alter the Inflammatory Response in Leishmania amazonensis-Infected Macrophages
- Leishmania (Leishmania) amazonensis induces macrophage miR-294 and miR-721 expression and modulates infection by targeting NOS2 and L-arginine metabolism
- RNA-seq transcriptional profiling of Leishmania amazonensis reveals an arginase- dependent gene expression regulation
Informações sobre o DOI: 10.1111/jpi.12321 (Fonte: oaDOI API)
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