Mitochondrial ATP-sensitive potassium channel opening inhibits isoproterenol-induced cardiac hypertrophy by preventing oxidative damage (2015)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1097/fjc.0000000000000210
- Subjects: ESTRESSE OXIDATIVO; MITOCÔNDRIAS; ANTIOXIDANTES
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2015
- Source:
- Título do periódico: Journal of Cardiovascular Pharmacology
- ISSN: 0160-2446
- Volume/Número/Paginação/Ano: v. 65, n. 4, p. 393-397, 2015
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
CALDAS, Francisco Rodrigo Lemos et al. Mitochondrial ATP-sensitive potassium channel opening inhibits isoproterenol-induced cardiac hypertrophy by preventing oxidative damage. Journal of Cardiovascular Pharmacology, v. 65, n. 4, p. 393-397, 2015Tradução . . Disponível em: https://doi.org/10.1097/fjc.0000000000000210. Acesso em: 19 abr. 2024. -
APA
Caldas, F. R. L., Leite, I. M. R., Filgueiras, A. B. T., Figueiredo Junior, I. L. de, Sousa, T. A. G. M. de, Martins, P. R., et al. (2015). Mitochondrial ATP-sensitive potassium channel opening inhibits isoproterenol-induced cardiac hypertrophy by preventing oxidative damage. Journal of Cardiovascular Pharmacology, 65( 4), 393-397. doi:10.1097/fjc.0000000000000210 -
NLM
Caldas FRL, Leite IMR, Filgueiras ABT, Figueiredo Junior IL de, Sousa TAGM de, Martins PR, Kowaltowski AJ, Facundo HDTF. Mitochondrial ATP-sensitive potassium channel opening inhibits isoproterenol-induced cardiac hypertrophy by preventing oxidative damage [Internet]. Journal of Cardiovascular Pharmacology. 2015 ; 65( 4): 393-397.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1097/fjc.0000000000000210 -
Vancouver
Caldas FRL, Leite IMR, Filgueiras ABT, Figueiredo Junior IL de, Sousa TAGM de, Martins PR, Kowaltowski AJ, Facundo HDTF. Mitochondrial ATP-sensitive potassium channel opening inhibits isoproterenol-induced cardiac hypertrophy by preventing oxidative damage [Internet]. Journal of Cardiovascular Pharmacology. 2015 ; 65( 4): 393-397.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1097/fjc.0000000000000210 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1097/fjc.0000000000000210 (Fonte: oaDOI API)
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