Omega-3 fatty acids protect from diet-induced obesity, glucose intolerance, and adipose tissue inflammation through PPARγ-dependent and PPARγ-independent actions (2015)
- Authors:
- Belchior, Thiago
- Paschoal, Vivian Almeida
- Magdalon, Juliana
- Chimin, Patricia
- Farias, Talita da Silva Mendes de
- Chaves Filho, Adriano de Britto
- Gorjão, Renata - Universidade Cruzeiro do Sul (UNICSUL)
- St-Pierre, Philippe - Université Laval (UL)
- Miyamoto, Sayuri

- Kang, Jing X
- Deshaies, Yves - Université Laval (UL)
- Marette, André - Université Laval (UL)
- Festuccia, William Tadeu Lara

- USP affiliated authors: MIYAMOTO, SAYURI - IQ ; FESTUCCIA, WILLIAM TADEU LARA - ICB
- Unidades: IQ; ICB
- DOI: 10.1002/mnfr.201400914
- Subjects: ÁCIDOS GRAXOS OMEGA 3; INFLAMAÇÃO
- Language: Inglês
- Imprenta:
- Source:
- Título: Molecular Nutrition Food Research
- ISSN: 1613-4125
- Volume/Número/Paginação/Ano: v. 59, p. 957-967, 2015
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
BELCHIOR, Thiago et al. Omega-3 fatty acids protect from diet-induced obesity, glucose intolerance, and adipose tissue inflammation through PPARγ-dependent and PPARγ-independent actions. Molecular Nutrition Food Research, v. 59, p. 957-967, 2015Tradução . . Disponível em: https://doi.org/10.1002/mnfr.201400914. Acesso em: 31 mar. 2026. -
APA
Belchior, T., Paschoal, V. A., Magdalon, J., Chimin, P., Farias, T. da S. M. de, Chaves Filho, A. de B., et al. (2015). Omega-3 fatty acids protect from diet-induced obesity, glucose intolerance, and adipose tissue inflammation through PPARγ-dependent and PPARγ-independent actions. Molecular Nutrition Food Research, 59, 957-967. doi:10.1002/mnfr.201400914 -
NLM
Belchior T, Paschoal VA, Magdalon J, Chimin P, Farias T da SM de, Chaves Filho A de B, Gorjão R, St-Pierre P, Miyamoto S, Kang JX, Deshaies Y, Marette A, Festuccia WTL. Omega-3 fatty acids protect from diet-induced obesity, glucose intolerance, and adipose tissue inflammation through PPARγ-dependent and PPARγ-independent actions [Internet]. Molecular Nutrition Food Research. 2015 ; 59 957-967.[citado 2026 mar. 31 ] Available from: https://doi.org/10.1002/mnfr.201400914 -
Vancouver
Belchior T, Paschoal VA, Magdalon J, Chimin P, Farias T da SM de, Chaves Filho A de B, Gorjão R, St-Pierre P, Miyamoto S, Kang JX, Deshaies Y, Marette A, Festuccia WTL. Omega-3 fatty acids protect from diet-induced obesity, glucose intolerance, and adipose tissue inflammation through PPARγ-dependent and PPARγ-independent actions [Internet]. Molecular Nutrition Food Research. 2015 ; 59 957-967.[citado 2026 mar. 31 ] Available from: https://doi.org/10.1002/mnfr.201400914 - Adipocyte mTORC1 deficiency promotes adipose tissue inflammation and NLRP3 inflammasome activation via oxidative stress and de novo ceramide synthesis
- Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease
- Regulation of adiposity by mTORC1
- Peroxisome proliferator-activated receptor γ activation favours selective subcutaneous lipid deposition by coordinately regulating lipoprotein lipase modulators, fatty acid transporters and lipogenic enzymes
- mTORC1 is required for brown adipose tissue recruitment and metabolic adaptation to cold
- A comparative perspective on lipid storage in animals
- Major involvement of mTOR in the PPARγ -induced stimulation of adipose tissue lipid uptake and fat accretion
- Dynamic changes in DICER levels in adipose tissue control metabolic adaptations to exercise
- Critical review of beige adipocyte thermogenic activation and contributionto whole-body energy expenditure
- Regulation of adipocyte and macrophage functions by mTORC1 and 2 in metabolic diseases
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