In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux (2015)
- Authors:
- Machado-Lima, Adriana
- Iborra, Rodrigo T.
- Pinto, Raphael S.
- Castilho, Gabriela
- Sartori, Camila Holanda
- Oliveira, Erika R.
- Okuda, Ligia S.
- Nakandakare, Edna R.
- Giannella-Neto, Daniel
- Machado, Ubiratan Fabres
- Corrêa-Giannella, Maria Lucia Cardillo
- Traldi, Pietro
- Porcu, Simona
- Roverso, Marco
- Lapolla, Annunziata
- Passarelli, Marisa
- USP affiliated authors: NAKANDAKARE, EDNA REGINA - FM ; GIANNELLA, MARIA LUCIA CARDILLO CORREA - FM ; MACHADO, UBIRATAN FABRES - ICB ; PASSARELLI, MARISA - FM
- Unidades: FM; ICB
- DOI: 10.1002/jcp.24860
- Subjects: COLESTEROL; MACRÓFAGOS; DIABETES MELLITUS NÃO INSULINO-DEPENDENTE; EXPRESSÃO GÊNICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Cellular Physiology
- ISSN: 0021-9541
- Volume/Número/Paginação/Ano: v. 230, n. 6, p. 1250-1257, 2015
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
MACHADO-LIMA, Adriana et al. In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux. Journal of Cellular Physiology, v. 230, n. 6, p. 1250-1257, 2015Tradução . . Disponível em: http://onlinelibrary.wiley.com/doi/10.1002/jcp.24860/epdf. Acesso em: 27 dez. 2025. -
APA
Machado-Lima, A., Iborra, R. T., Pinto, R. S., Castilho, G., Sartori, C. H., Oliveira, E. R., et al. (2015). In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux. Journal of Cellular Physiology, 230( 6), 1250-1257. doi:10.1002/jcp.24860 -
NLM
Machado-Lima A, Iborra RT, Pinto RS, Castilho G, Sartori CH, Oliveira ER, Okuda LS, Nakandakare ER, Giannella-Neto D, Machado UF, Corrêa-Giannella MLC, Traldi P, Porcu S, Roverso M, Lapolla A, Passarelli M. In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux [Internet]. Journal of Cellular Physiology. 2015 ; 230( 6): 1250-1257.[citado 2025 dez. 27 ] Available from: http://onlinelibrary.wiley.com/doi/10.1002/jcp.24860/epdf -
Vancouver
Machado-Lima A, Iborra RT, Pinto RS, Castilho G, Sartori CH, Oliveira ER, Okuda LS, Nakandakare ER, Giannella-Neto D, Machado UF, Corrêa-Giannella MLC, Traldi P, Porcu S, Roverso M, Lapolla A, Passarelli M. In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux [Internet]. Journal of Cellular Physiology. 2015 ; 230( 6): 1250-1257.[citado 2025 dez. 27 ] Available from: http://onlinelibrary.wiley.com/doi/10.1002/jcp.24860/epdf - RAGE mediates cholesterol efflux impairment in macrophages caused by human advanced glycated albumin
- AGE-albumin enhances ABCA1 degradation by ubiquitin-proteasome and lysosomal pathways in macrophages
- Aerobic exercise training enhances the in vivo cholesterol trafficking from macrophages to the liver independently of changes in the expression of genes involved in lipid flux in macrophages and aorta
- The reverse cholesterol transport is impaired by serum albumin isolated from uncontrolled diabetes mellitus patientes
- Oleic and linoleic fatty acids downregulate Slc2a4/GLUT4 expression via NFKB and SREBP1 in skeletal muscle cells
- Selective inhibition of proteasomal and lysosomal degradation pathways partially prevent abca-1 reduction in macrophages induced by advanced glycated albumin
- Advanced glycated albumin isolated from poorly controlled type 1 diabetes mellitus patients alters macrophage gene expression impairing ABCA-1-mediated reverse cholesterol transport
- Endoplasmic reticulum stress inhibition prevents ABCA-1 reductio in glycated albumin-treated macrophages
- ER stress is associated with reduced ABCA-1 protein levels in macrophages treated with advanced glycated albumin – Reversal by a chemical chaperone
- Glicação avançada altera a incorporação de ácido oléico em lípides e reduz síntese de colesterol em macrófagos
Informações sobre o DOI: 10.1002/jcp.24860 (Fonte: oaDOI API)
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