IKKβ inhibition suppresses sphere formation and self renewal of lung cancer initiating cells (2014)
- Authors:
- Autor USP: BASSÈRES, DANIELA SANCHEZ - IQ
- Unidade: IQ
- Subjects: ONCOGENES; NEOPLASIAS DO TRATO RESPIRATÓRIO
- Language: Inglês
- Imprenta:
- Source:
- Título: European Journal of Cancer
- ISSN: 0959-8049
- Volume/Número/Paginação/Ano: v. 50, suppl. 6, p. 103 res. 317, 2014
- Conference titles: EORTC - NCI - AACR Symposium on Molecular Targets and Cancer Therapeutics
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ABNT
CARNEIRO LOBO, Tatiana Correa et al. IKKβ inhibition suppresses sphere formation and self renewal of lung cancer initiating cells. European Journal of Cancer. Oxford: Instituto de Química, Universidade de São Paulo. . Acesso em: 29 dez. 2025. , 2014 -
APA
Carneiro Lobo, T. C., Salviatto, A. P., Baldwin, A. S., & Bassères, D. S. (2014). IKKβ inhibition suppresses sphere formation and self renewal of lung cancer initiating cells. European Journal of Cancer. Oxford: Instituto de Química, Universidade de São Paulo. -
NLM
Carneiro Lobo TC, Salviatto AP, Baldwin AS, Bassères DS. IKKβ inhibition suppresses sphere formation and self renewal of lung cancer initiating cells. European Journal of Cancer. 2014 ; 50 103 res. 317.[citado 2025 dez. 29 ] -
Vancouver
Carneiro Lobo TC, Salviatto AP, Baldwin AS, Bassères DS. IKKβ inhibition suppresses sphere formation and self renewal of lung cancer initiating cells. European Journal of Cancer. 2014 ; 50 103 res. 317.[citado 2025 dez. 29 ] - Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer
- Targeted BMI1 inhibition impairs tumor growth in lung adenocarcinomas with low CEBP alpha expression
- Exploring IKKβ kinase as a therapeutic target for KRAS-driven lung tumor initiating cells
- Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer
- Targeting Aurora A kinase activity in lung cancer initiating cells driven by oncogenic KRAS
- Aurora kinase targeting in lung cancer reduces KRAS-induced transformation
- Tumor microenvironment landscapes supporting EGFR-mutant NSCLC are modulated at the single-cell interaction level by unesbulin treatment
- IKKβ kinase promotes stemness, migration, and invasion in KRAS-driven lung adenocarcinoma cells
- Identification of a targetable KRAS-mutant epithelial population in non-small cell lung cancer
- The IKK'beta' kinase is a potential therapeutic target in K-Ras-induced lung cancer
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