Analysis of differentially expressed genes in a mouse model of KRAS-induced lung cancer with conditional loss of thep65 subunit of the NF-kBtranscription factor (2014)
- Authors:
- USP affiliated authors: ALMEIDA, SERGIO VERJOVSKI DE - IQ ; BASSÈRES, DANIELA SANCHEZ - IQ
- Unidade: IQ
- Subjects: EXPRESSÃO GÊNICA; NEOPLASIAS DO TRATO RESPIRATÓRIO
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Biologia Celular/SBBC
- Publisher place: São Paulo
- Date published: 2014
- Source:
- Título do periódico: Poster Abstracts
- Conference titles: Meeting of the Brazilian Society for Cell Biology
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ABNT
CORRÊA, Thalita Bueno et al. Analysis of differentially expressed genes in a mouse model of KRAS-induced lung cancer with conditional loss of thep65 subunit of the NF-kBtranscription factor. 2014, Anais.. São Paulo: Sociedade Brasileira de Biologia Celular/SBBC, 2014. . Acesso em: 28 mar. 2024. -
APA
Corrêa, T. B., Salviatto, A. P., Beckedorff, F. C. F., Ebbs, A., Verjovski-Almeida, S., Baldwin, A. S., & Bassères, D. S. (2014). Analysis of differentially expressed genes in a mouse model of KRAS-induced lung cancer with conditional loss of thep65 subunit of the NF-kBtranscription factor. In Poster Abstracts. São Paulo: Sociedade Brasileira de Biologia Celular/SBBC. -
NLM
Corrêa TB, Salviatto AP, Beckedorff FCF, Ebbs A, Verjovski-Almeida S, Baldwin AS, Bassères DS. Analysis of differentially expressed genes in a mouse model of KRAS-induced lung cancer with conditional loss of thep65 subunit of the NF-kBtranscription factor. Poster Abstracts. 2014 ;[citado 2024 mar. 28 ] -
Vancouver
Corrêa TB, Salviatto AP, Beckedorff FCF, Ebbs A, Verjovski-Almeida S, Baldwin AS, Bassères DS. Analysis of differentially expressed genes in a mouse model of KRAS-induced lung cancer with conditional loss of thep65 subunit of the NF-kBtranscription factor. Poster Abstracts. 2014 ;[citado 2024 mar. 28 ] - Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer
- Targeted BMI1 inhibition impairs tumor growth in lung adenocarcinomas with low CEBP alpha expression
- FOXO1 inhibition by oncogenic KRAS as an important mechanism promoting lung oncogenesis
- Requirement of the NF-'capa' B subunit p65/RelA for 'capa'-Ras-induced lung tumorigenesis
- Resenha sem título próprio
- Targeting Aurora kinase activity in lung cancer cell driven by oncogenic KRAS impairs lung cancer initiating cell function
- Exploring IKKβ as an anti-angiogenic therapeutic target in KRAS-induced lung cancer
- IKK is a therapeutic target in KRAS-induced lung cancer with disrupted p53 activity
- Exploring IKKβ kinase as a therapeutic target for KRAS-driven lung tumor initiating cells
- Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer
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