Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats (2014)
- Authors:
- USP affiliated authors: GRANDO, MARCELLA DARUGE - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- DOI: 10.1152/ajpheart.00520.2013
- Subjects: ÓXIDO NÍTRICO; ENDOTÉLIO; GLUTAMATOS
- Language: Inglês
- Imprenta:
- Source:
- Título: American Journal of Physiology Heart and Circulatory Physiology
- ISSN: 0363-6135
- Volume/Número/Paginação/Ano: v. 306, n. 10, p. H1472-H1480, 2014
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
- Licença: other-oa
-
ABNT
CUNHA, Natalia Veronez da et al. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats. American Journal of Physiology Heart and Circulatory Physiology, v. 306, n. 10, p. H1472-H1480, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00520.2013. Acesso em: 29 dez. 2025. -
APA
Cunha, N. V. da, Pinge-Filho, P., Panis, C., Silva, B. R., Pernomian, L., Grando, M. D., et al. (2014). Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats. American Journal of Physiology Heart and Circulatory Physiology, 306( 10), H1472-H1480. doi:10.1152/ajpheart.00520.2013 -
NLM
Cunha NV da, Pinge-Filho P, Panis C, Silva BR, Pernomian L, Grando MD, Cecchini R, Bendhack LM, Martins-Pinge MC. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats [Internet]. American Journal of Physiology Heart and Circulatory Physiology. 2014 ; 306( 10): H1472-H1480.[citado 2025 dez. 29 ] Available from: https://doi.org/10.1152/ajpheart.00520.2013 -
Vancouver
Cunha NV da, Pinge-Filho P, Panis C, Silva BR, Pernomian L, Grando MD, Cecchini R, Bendhack LM, Martins-Pinge MC. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats [Internet]. American Journal of Physiology Heart and Circulatory Physiology. 2014 ; 306( 10): H1472-H1480.[citado 2025 dez. 29 ] Available from: https://doi.org/10.1152/ajpheart.00520.2013 - Endothelial cyclooxygenase differently modulates the contractile response to phenylephrine but not to thromboxane analog in aorta from normotensive and hypertensive rats
- Increased resting tension abolishes the endothelial anti-contractile effect induced by phenylephrine in renal hypertensive (2K-1C) rat aorta
- Inibição da NO-sintase normaliza a resposta contrátil estimulada com fenilefrina em aorta de ratos hipertensos renais 2R-1C
- Fenilefrina ativa a enzima NO-sintase em aorta de ratos heipertensos via AKT e fosforilação da serina1177
- Hydrogen peroxide modulates the contractile response induced by phenylephrine in renal hypertensive rat aorta
- Phenylephrine activates eNOS Ser1177 phosphorylation and nitric oxide signaling in renal hypertensive rat aorta
- Vascular relaxation induced by a nitric oxide donor and cyclooxygenase inhibitor in hypertension
- Endothelial nitric oxide synthase and cyclooxygenase are activated by hydrogen peroxide in renal hypertensive rat aorta
- Testosterone increases bradykinin-induced relaxation in the coronary bed of hypertensive rats
- Peróxido de hidrogênio reduz a resposta contrátil para fenilefrina em aorta de ratos hipertensos renais
Informações sobre o DOI: 10.1152/ajpheart.00520.2013 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
