TGF-Beta in the pathogenesis of glomerulonephritis in experimental leishamaniasis (2007)
- Authors:
- USP affiliated authors: GOTO, HIRO - FM
- Unidades: FM
- Subjects: GLOMERUNEFRITE; MODELOS ANIMAIS; NEFROPATIAS EM ANIMAL
- Language: Inglês
- Imprenta:
- Publisher: Society of Protozoology
- Place of publication: Caxambu
- Date published: 2007
- Source:
- Título do periódico: Resumos
- Volume/Número/Paginação/Ano: p. 150, res. IM68, 2007
- Conference title: Annual Meeting of the Brazilian Society of Protozoology
-
ABNT
PRIANTI, M. G. P.; CASTRO, L. R. de; CARVALHO, M. D. T.; GOTO, Hiro. TGF-Beta in the pathogenesis of glomerulonephritis in experimental leishamaniasis. Anais.. Caxambu: Society of Protozoology, 2007. -
APA
Prianti, M. G. P., Castro, L. R. de, Carvalho, M. D. T., & Goto, H. (2007). TGF-Beta in the pathogenesis of glomerulonephritis in experimental leishamaniasis. In Resumos (p. 150). Caxambu: Society of Protozoology. -
NLM
Prianti MGP, Castro LR de, Carvalho MDT, Goto H. TGF-Beta in the pathogenesis of glomerulonephritis in experimental leishamaniasis. Resumos. 2007 ; 150. -
Vancouver
Prianti MGP, Castro LR de, Carvalho MDT, Goto H. TGF-Beta in the pathogenesis of glomerulonephritis in experimental leishamaniasis. Resumos. 2007 ; 150. - Apoptose em leishmaniose cutânea humana
- Fator de crescimento humano insulina-símile
- Participation of immnunoglobulins in the pathogenesis of inflammation in liver in visceral leishmaniasis
- Effect of insulin-like growth factor (IGF) I and II on the nitric oxide (NO) production by Leishmania-Infected macrophages
- Immunopathogenesis of experimental visceral Leishmaniasis (VL)
- CD4(+) T cells alter the stromal microenvironment and repress medullary erythropoiesis in murine visceral leishmaniasis
- Neutrophils participate in the evolution of leishmania (L) chagasi infection in mice
- Tegumentary leishmaniasis as a manifestation of immune reconstitution inflammatory syndrome in 2 patients with AIDS
- Alterações imunopatológicas na co-infecção HIV-Leishmania
- Participation of immunoglobulins and cells in the pathogenesis of glomerulonephritis in visceral leishmaniasis
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