Polo-like kinase 1 inhibition causes decreased proliferation by cell cycle arrest, leading to cell death in glioblastoma (2013)
- Authors:
- USP affiliated authors: MORALES, ANDRESSA GOIS - IB ; QUEIROZ, ROSANE GOMES DE PAULA - FMRP ; MACHADO, HÉLIO RUBENS - FMRP ; CARLOTTI JUNIOR, CARLOS GILBERTO - FMRP ; SERAFINI, LUCIANO NEDER - FMRP ; SCRIDELI, CARLOS ALBERTO - FMRP ; TONE, LUIZ GONZAGA - FMRP
- Unidades: IB; FMRP
- DOI: 10.1038/cgt.2013.46
- Subjects: ASTROCITOMA; PROTEÍNAS QUINASES; CICLO CELULAR; INIBIDORES DE ENZIMAS
- Language: Inglês
- Imprenta:
- Source:
- Título: Cancer Gene Therapy
- ISSN: 0929-1903
- Volume/Número/Paginação/Ano: v. 20, n. 9, p. 499-506, 2013
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão submetida (Pré-print)
- Acessar versão aberta:
-
ABNT
PEZUK, J. A. et al. Polo-like kinase 1 inhibition causes decreased proliferation by cell cycle arrest, leading to cell death in glioblastoma. Cancer Gene Therapy, v. 20, n. 9, p. 499-506, 2013Tradução . . Disponível em: https://doi.org/10.1038/cgt.2013.46. Acesso em: 01 abr. 2026. -
APA
Pezuk, J. A., Brassesco, M. S., Morales, A. G., Oliveira, J. C. de, Queiroz, R. G. de P., Machado, H. R., et al. (2013). Polo-like kinase 1 inhibition causes decreased proliferation by cell cycle arrest, leading to cell death in glioblastoma. Cancer Gene Therapy, 20( 9), 499-506. doi:10.1038/cgt.2013.46 -
NLM
Pezuk JA, Brassesco MS, Morales AG, Oliveira JC de, Queiroz RG de P, Machado HR, Carlotti Júnior CG, Serafini LN, Scrideli CA, Tone LG. Polo-like kinase 1 inhibition causes decreased proliferation by cell cycle arrest, leading to cell death in glioblastoma [Internet]. Cancer Gene Therapy. 2013 ; 20( 9): 499-506.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1038/cgt.2013.46 -
Vancouver
Pezuk JA, Brassesco MS, Morales AG, Oliveira JC de, Queiroz RG de P, Machado HR, Carlotti Júnior CG, Serafini LN, Scrideli CA, Tone LG. Polo-like kinase 1 inhibition causes decreased proliferation by cell cycle arrest, leading to cell death in glioblastoma [Internet]. Cancer Gene Therapy. 2013 ; 20( 9): 499-506.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1038/cgt.2013.46 - Differential expression of 12 histone deacetylase (HDAC) genes in astrocytomas and normal brain tissue: class II and IV are hypoexpressed in glioblastomas
- BUB1 and BUBR1 inhibition decreases proliferation and colony formation, and enhances radiation sensitivity in pediatric glioblastoma cells
- Low MIR-196B and high MIR-708 expression are associated with T-cell childhood acute lymphoblastic leukemia
- PLK1 expression and Bl 2536 effects in childhood acute lymphoblastic leukemia
- Silencing of checkpoint mitotic genes inhibits the proliferation and clonogenicity in pediatric glioblastoma cell line
- Effects of polo-like kinase 1 inhibition on glioblastoma cell lines
- Quantitative PCR analysis of the expression profile of genes related to multiple drug resistance in tumors of the central nervous system
- Abnormal methylation of histone deacetylase genes: implications on etiology and epigenetic therapy of astrocytomas
- Differential expression of migration and adhesion genes among neuroepithelial tumors of children and adults
- Expression profile of the hypoxia-related genes CA9, CA12, VEGF, SLC2A1, HIF-1A and HIF-1A and HIF-2A in central nervous system tumors (CNS)
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