Increased endogenous hydrogen sulfide formation contributes to inflammation and epithelial-mesenchymal transition in the renal damage induced by cisplatin (2013)
- Authors:
- USP affiliated authors: COIMBRA, TEREZILA MACHADO - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; COSTA, ROBERTO SILVA - FMRP
- Unidade: FMRP
- Subjects: NEFROPATIAS; TRANSPLANTE DE RIM
- Language: Inglês
- Imprenta:
- Source:
- Título: Abstracts
- Conference titles: World Congress of Nephrology
-
ABNT
FRANCESCATO, Heloísa Della Coletta et al. Increased endogenous hydrogen sulfide formation contributes to inflammation and epithelial-mesenchymal transition in the renal damage induced by cisplatin. 2013, Anais.. Hong Kong: ISN, 2013. . Acesso em: 01 jan. 2026. -
APA
Francescato, H. D. C., Cunha, F. de Q., Silva, C. da, Bertozi, G., Costa, R. S., & Coimbra, T. M. (2013). Increased endogenous hydrogen sulfide formation contributes to inflammation and epithelial-mesenchymal transition in the renal damage induced by cisplatin. In Abstracts. Hong Kong: ISN. -
NLM
Francescato HDC, Cunha F de Q, Silva C da, Bertozi G, Costa RS, Coimbra TM. Increased endogenous hydrogen sulfide formation contributes to inflammation and epithelial-mesenchymal transition in the renal damage induced by cisplatin. Abstracts. 2013 ;[citado 2026 jan. 01 ] -
Vancouver
Francescato HDC, Cunha F de Q, Silva C da, Bertozi G, Costa RS, Coimbra TM. Increased endogenous hydrogen sulfide formation contributes to inflammation and epithelial-mesenchymal transition in the renal damage induced by cisplatin. Abstracts. 2013 ;[citado 2026 jan. 01 ] - ATP-sensitive potassium channels blockage attenuates cisplatin-induced renal damage
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