Medial amygdaloid nucleus 5-H'T IND.2c' receptors are involved in the hypophagic effect caused by zimelidine in rats (2012)
- Authors:
- USP affiliated authors: CORREA, FERNANDO MORGAN DE AGUIAR - FMRP ; MORAES, LEONARDO RESSTEL BARBOSA - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.neuropharm.2012.03.020
- Subjects: COMPORTAMENTO ALIMENTAR (FISIOLOGIA); RECEPTORES DE SEROTONINA; AMÍGDALA DO CEREBELO (EFEITOS DE DROGAS)
- Language: Inglês
- Imprenta:
- Publisher place: Kidlington
- Date published: 2012
- Source:
- Título: Neuropharmacology
- ISSN: 0028-3908
- Volume/Número/Paginação/Ano: v. 63, n. 2, p. 301-309, 2012
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
SCOPINHO, A. A. et al. Medial amygdaloid nucleus 5-H'T IND.2c' receptors are involved in the hypophagic effect caused by zimelidine in rats. Neuropharmacology, v. 63, n. 2, p. 301-309, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2012.03.020. Acesso em: 23 fev. 2026. -
APA
Scopinho, A. A., Fortaleza, E. A. T., Corrêa, F. M. de A., & Resstel, L. B. M. (2012). Medial amygdaloid nucleus 5-H'T IND.2c' receptors are involved in the hypophagic effect caused by zimelidine in rats. Neuropharmacology, 63( 2), 301-309. doi:10.1016/j.neuropharm.2012.03.020 -
NLM
Scopinho AA, Fortaleza EAT, Corrêa FM de A, Resstel LBM. Medial amygdaloid nucleus 5-H'T IND.2c' receptors are involved in the hypophagic effect caused by zimelidine in rats [Internet]. Neuropharmacology. 2012 ; 63( 2): 301-309.[citado 2026 fev. 23 ] Available from: https://doi.org/10.1016/j.neuropharm.2012.03.020 -
Vancouver
Scopinho AA, Fortaleza EAT, Corrêa FM de A, Resstel LBM. Medial amygdaloid nucleus 5-H'T IND.2c' receptors are involved in the hypophagic effect caused by zimelidine in rats [Internet]. Neuropharmacology. 2012 ; 63( 2): 301-309.[citado 2026 fev. 23 ] Available from: https://doi.org/10.1016/j.neuropharm.2012.03.020 - Non-N-methyl-D-aspartate glutamate receptors in the lateral hypothalamus modulate cardiac baroreflex responses in conscious rats
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Informações sobre o DOI: 10.1016/j.neuropharm.2012.03.020 (Fonte: oaDOI API)
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