Expression of IGF2 and IGF1R in pediatric adrenocortical tumors (ACT): overexpression of IGF1R is associated with unfavorable event and metastasis in adrenocortical carcinomas (2012)
- Authors:
- Lira, R. C. P.
- Fedatto, P. F.
- Martinelli Junior, Carlos Eduardo
- Antonini, Sonir Roberto Rauber
- Castro, Margaret de
- Tucci Júnior, Silvio
- Neder, Luciano
- Ramalho, Leandra Naíra Zambelli
- Seidinger, A. L.
- Cardinalli, I.
- Mastellaro, M. J.
- Yunes, J. A.
- Brandalise, Silvia Regina
- Tone, Luiz Gonzaga
- Scrideli, Carlos Alberto
- USP affiliated authors: MARTINELLI JUNIOR, CARLOS EDUARDO - FMRP ; ANTONINI, SONIR ROBERTO RAUBER - FMRP ; CASTRO, MARGARET DE - FMRP ; TUCCI JUNIOR, SILVIO - FMRP ; SERAFINI, LUCIANO NEDER - FMRP ; RAMALHO, LEANDRA NAIRA ZAMBELLI - FMRP ; TONE, LUIZ GONZAGA - FMRP ; SCRIDELI, CARLOS ALBERTO - FMRP
- Unidade: FMRP
- Subjects: NEOPLASIAS; PEDIATRIA; EXPRESSÃO GÊNICA; CRIANÇAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Growth Hormone and IGF Research
- ISSN: 1096-6374
- Volume/Número/Paginação/Ano: v. 22, suppl. 1, p. S41, res. P01-27, 2012
-
ABNT
LIRA, R. C. P. et al. Expression of IGF2 and IGF1R in pediatric adrenocortical tumors (ACT): overexpression of IGF1R is associated with unfavorable event and metastasis in adrenocortical carcinomas. Growth Hormone and IGF Research. London: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 25 abr. 2024. , 2012 -
APA
Lira, R. C. P., Fedatto, P. F., Martinelli Junior, C. E., Antonini, S. R. R., Castro, M. de, Tucci Júnior, S., et al. (2012). Expression of IGF2 and IGF1R in pediatric adrenocortical tumors (ACT): overexpression of IGF1R is associated with unfavorable event and metastasis in adrenocortical carcinomas. Growth Hormone and IGF Research. London: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Lira RCP, Fedatto PF, Martinelli Junior CE, Antonini SRR, Castro M de, Tucci Júnior S, Neder L, Ramalho LNZ, Seidinger AL, Cardinalli I, Mastellaro MJ, Yunes JA, Brandalise SR, Tone LG, Scrideli CA. Expression of IGF2 and IGF1R in pediatric adrenocortical tumors (ACT): overexpression of IGF1R is associated with unfavorable event and metastasis in adrenocortical carcinomas. Growth Hormone and IGF Research. 2012 ; 22 S41.[citado 2024 abr. 25 ] -
Vancouver
Lira RCP, Fedatto PF, Martinelli Junior CE, Antonini SRR, Castro M de, Tucci Júnior S, Neder L, Ramalho LNZ, Seidinger AL, Cardinalli I, Mastellaro MJ, Yunes JA, Brandalise SR, Tone LG, Scrideli CA. Expression of IGF2 and IGF1R in pediatric adrenocortical tumors (ACT): overexpression of IGF1R is associated with unfavorable event and metastasis in adrenocortical carcinomas. Growth Hormone and IGF Research. 2012 ; 22 S41.[citado 2024 abr. 25 ] - Low expression of HLA-DRA, HLA-DPA1, and HLA-DPB1 is associated with poor prognosis in pediatric adrenocortical tumors (ACT)
- Associação da expressão do IGF1R com metástases de tumores adrenocorticais pediátricos e o papel do IGF1R na secreção de hormônios e sobrevivência das células de carcinoma adrenocortical (NCI-H295A)
- Overexpression of IGF1R but not IGF2 is associated with unfavorable event and metastasis in adrenocortical carcinomas
- Correlations between gene expression of IGF1R and IGF2 and WNT/B-catenin pathways in paediatric adrenocortical tumors (ACTs)
- Spindle assembly checkpoint gene expression in childhood adrenocortical tumors (ACT): overexpression of aurora kinases A and B is associated with a poor prognosis
- Expression profile of apoptosis-related genes in childhood adrenocortical tumors: low level of expression of BCL2 and TNF genes suggests a poor prognosis
- Correlations between IGF system and WNT/b-catenin pathway in pediatric adrenocortical tumors
- Class II histocompatibility antigens (HLA) in childhood adrenocortical tumors (ACT): low expression is related with poor prognosis
- Underexpression of class II histocompatibility antigens (HLA) is related with poor prognosis in childhood adrenocortical tumors (ACT)
- Functional inhibition of IGF1R in adrenocortical tumors cells promotes cellular death and changes genes expression of WNT/beta-catenin
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