Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65 (2012)
- Authors:
- Autor USP: NUNES, MARIA TEREZA - ICB
- Unidade: ICB
- Assunto: FISIOLOGIA HUMANA
- Language: Inglês
- Imprenta:
- Publisher: Dep/Physiol/Biophys/ICB/USP
- Publisher place: São Paulo
- Date published: 2012
- Source:
- Título: Abstracts
- Conference titles: Thematic Symposium: new frontiers in applied physiology
-
ABNT
SERRANO-NASCIMENTO, Caroline et al. Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65. 2012, Anais.. São Paulo: Dep/Physiol/Biophys/ICB/USP, 2012. . Acesso em: 15 fev. 2026. -
APA
Serrano-Nascimento, C., Nicola, J. P., Masini-Repiso, A. M., & Nunes, M. T. (2012). Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65. In Abstracts. São Paulo: Dep/Physiol/Biophys/ICB/USP. -
NLM
Serrano-Nascimento C, Nicola JP, Masini-Repiso AM, Nunes MT. Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65. Abstracts. 2012 ;[citado 2026 fev. 15 ] -
Vancouver
Serrano-Nascimento C, Nicola JP, Masini-Repiso AM, Nunes MT. Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65. Abstracts. 2012 ;[citado 2026 fev. 15 ] - Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood
- Iodide excess reduces the activity of sodium-iodide symporter (nis) promoter: the role of PAX8 and P65 transcription factors on the inhibitory effects of this oligoelementrats
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- Arginine induces GH gene expression by activating NOS/NO signaling in rat isolated hemi-pituitaries
- Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring
- Thyroid hormone treatment decreases hepatic glucose production and renal reabsorption of glucose in alloxan-induced diabetic Wistar rats
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