Effects fo inos inhibition on nnos expression in inflammatory and bronchial epithelial cells in guinea pigs with chronic lung inflammation (2005)
- Authors:
- USP affiliated authors: MALDONADO, EDNA APARECIDA LEICK - FM ; MARTINS, MILTON DE ARRUDA - FM
- Unidade: FM
- Subjects: INFLAMAÇÃO; CÉLULAS EPITELIAIS (EFEITOS); COBAIAS; DOENÇA CRÔNICA; PULMÃO (PATOLOGIA)
- Language: Inglês
- Imprenta:
- Source:
- Título: Resumos
- Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental
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ABNT
YANO, L et al. Effects fo inos inhibition on nnos expression in inflammatory and bronchial epithelial cells in guinea pigs with chronic lung inflammation. 2005, Anais.. São Paulo: FeSBE, 2005. . Acesso em: 25 fev. 2026. -
APA
Yano, L., Prado, C. M., Miyamoto, L., Maldonado, E. A. L., Martins, M. de A., & Tiberio, I. F. L. C. (2005). Effects fo inos inhibition on nnos expression in inflammatory and bronchial epithelial cells in guinea pigs with chronic lung inflammation. In Resumos. São Paulo: FeSBE. -
NLM
Yano L, Prado CM, Miyamoto L, Maldonado EAL, Martins M de A, Tiberio IFLC. Effects fo inos inhibition on nnos expression in inflammatory and bronchial epithelial cells in guinea pigs with chronic lung inflammation. Resumos. 2005 ;[citado 2026 fev. 25 ] -
Vancouver
Yano L, Prado CM, Miyamoto L, Maldonado EAL, Martins M de A, Tiberio IFLC. Effects fo inos inhibition on nnos expression in inflammatory and bronchial epithelial cells in guinea pigs with chronic lung inflammation. Resumos. 2005 ;[citado 2026 fev. 25 ] - Modulation of chronic airway inflammation by oral tolerance:: effects in lung mechanics, exhaled nitric oxide and airway hyperresponsiveness to methacholine in guinea pigs
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- Assessment of eosinophils in the bone marrow and airway wall of guinea pigs with chronic lung inflammation: modulation by different ways of oral induced tolerance
- Oral tolerance attenuates airway inflammation and remodeling in a model of chronic pulmonary allergic inflammation in guinea pigs
- Effects of chronic nitric oxide inhibition on eosinophil recruitment and extracellular matrix remodelling in chronic pulmonary inflammation
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