Eag1, Eag2, and SK3 potassium Channel expression in the rat hippocampus after global transient brain ischemia (2012)
- Authors:
- USP affiliated authors: JOCA, SÂMIA REGIANE LOURENÇO - FCFRP ; GUIMARAES, ELAINE APARECIDA DEL BEL BELLUZ - FORP
- Unidades: FCFRP; FORP
- DOI: 10.1002/jnr.22772
- Subjects: ISQUEMIA CEREBRAL (METABOLISMO;FISIOPATOLOGIA); COMPORTAMENTO ANIMAL (FISIOLOGIA); NEURÔNIOS
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Neuroscience Research
- ISSN: 0360-4012
- Volume/Número/Paginação/Ano: v. 90, n. 3, p. 632-640, 2012
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
OLIVEIRA, R. M. Weffort de et al. Eag1, Eag2, and SK3 potassium Channel expression in the rat hippocampus after global transient brain ischemia. Journal of Neuroscience Research, v. 90, n. 3, p. 632-640, 2012Tradução . . Disponível em: https://doi.org/10.1002/jnr.22772. Acesso em: 29 dez. 2025. -
APA
Oliveira, R. M. W. de, Martin, S., Oliveira, C. L. de, Milani, H., Schiavon, A. P., Joca, S. R. L., et al. (2012). Eag1, Eag2, and SK3 potassium Channel expression in the rat hippocampus after global transient brain ischemia. Journal of Neuroscience Research, 90( 3), 632-640. doi:10.1002/jnr.22772 -
NLM
Oliveira RMW de, Martin S, Oliveira CL de, Milani H, Schiavon AP, Joca SRL, Pardo LA, Stühmer W, Del Bel EA. Eag1, Eag2, and SK3 potassium Channel expression in the rat hippocampus after global transient brain ischemia [Internet]. Journal of Neuroscience Research. 2012 ; 90( 3): 632-640.[citado 2025 dez. 29 ] Available from: https://doi.org/10.1002/jnr.22772 -
Vancouver
Oliveira RMW de, Martin S, Oliveira CL de, Milani H, Schiavon AP, Joca SRL, Pardo LA, Stühmer W, Del Bel EA. Eag1, Eag2, and SK3 potassium Channel expression in the rat hippocampus after global transient brain ischemia [Internet]. Journal of Neuroscience Research. 2012 ; 90( 3): 632-640.[citado 2025 dez. 29 ] Available from: https://doi.org/10.1002/jnr.22772 - Tolerance to the cataleptic effect that follows repeated nitric oxide synthase inhibition may be related to functional enzymatic recovery
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Informações sobre o DOI: 10.1002/jnr.22772 (Fonte: oaDOI API)
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