Mechanisms of manganese-induced neurotoxicity in primary neuronal cultures: the role of manganese speciation and cell type (2011)
- Authors:
- USP affiliated authors: ESPOSITO, BRENO PANNIA - IQ ; LARDNER, NADJA CRISTHINA DE SOUZA PINTO - IQ ; BARBOSA JUNIOR, FERNANDO - FCFRP
- Unidades: IQ; FCFRP
- DOI: 10.1093/toxsci/kfr234
- Subjects: MANGANÊS; NEUROQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Toxicological Sciences
- ISSN: 1096-6080
- Volume/Número/Paginação/Ano: v. 124, n. 2, p. 414-423, 2011
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
HERNÁNDEZ, Raúl Bonne et al. Mechanisms of manganese-induced neurotoxicity in primary neuronal cultures: the role of manganese speciation and cell type. Toxicological Sciences, v. 124, n. 2, p. 414-423, 2011Tradução . . Disponível em: https://doi.org/10.1093/toxsci/kfr234. Acesso em: 19 abr. 2024. -
APA
Hernández, R. B., Farina, M., Espósito, B. P., Souza-Pinto, N. C. de, Barbosa Júnior, F., & Suñol, C. (2011). Mechanisms of manganese-induced neurotoxicity in primary neuronal cultures: the role of manganese speciation and cell type. Toxicological Sciences, 124( 2), 414-423. doi:10.1093/toxsci/kfr234 -
NLM
Hernández RB, Farina M, Espósito BP, Souza-Pinto NC de, Barbosa Júnior F, Suñol C. Mechanisms of manganese-induced neurotoxicity in primary neuronal cultures: the role of manganese speciation and cell type [Internet]. Toxicological Sciences. 2011 ; 124( 2): 414-423.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/toxsci/kfr234 -
Vancouver
Hernández RB, Farina M, Espósito BP, Souza-Pinto NC de, Barbosa Júnior F, Suñol C. Mechanisms of manganese-induced neurotoxicity in primary neuronal cultures: the role of manganese speciation and cell type [Internet]. Toxicological Sciences. 2011 ; 124( 2): 414-423.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/toxsci/kfr234 - Manganese-induced development neurotoxicity is mediated by chemical speciation and probably by mitochondrial impairment
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Informações sobre o DOI: 10.1093/toxsci/kfr234 (Fonte: oaDOI API)
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