Effects of statins on matrix metalloproteinases and their endogenous inhibitors in human endothelial cells (2011)
- Authors:
- USP affiliated authors: GERLACH, RAQUEL FERNANDA - FORP ; SANTOS, JOSE EDUARDO TANUS DOS - FMRP
- Unidades: FORP; FMRP
- DOI: 10.1007/s00210-011-0623-0
- Subjects: CÉLULAS ENDOTELIAIS; METALOPROTEINASES
- Language: Inglês
- Imprenta:
- Publisher place: Heidelberg
- Date published: 2011
- Source:
- Título: Naunyn-Schmiedeberg's Archives of Pharmacology
- ISSN: 0028-1298
- Volume/Número/Paginação/Ano: v. 383, n. 6, p. 547-554, 2011
- Este artigo NÃO possui versão em acesso aberto
-
Status: Nenhuma versão em acesso aberto identificada -
ABNT
IZIDORO-TOLEDO, Tatiane C. et al. Effects of statins on matrix metalloproteinases and their endogenous inhibitors in human endothelial cells. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 383, n. 6, p. 547-554, 2011Tradução . . Disponível em: https://doi.org/10.1007/s00210-011-0623-0. Acesso em: 15 mar. 2026. -
APA
Izidoro-Toledo, T. C., Guimarães, D. A., Belo, V. A., Gerlach, R. F., & Tanus-Santos, J. E. (2011). Effects of statins on matrix metalloproteinases and their endogenous inhibitors in human endothelial cells. Naunyn-Schmiedeberg's Archives of Pharmacology, 383( 6), 547-554. doi:10.1007/s00210-011-0623-0 -
NLM
Izidoro-Toledo TC, Guimarães DA, Belo VA, Gerlach RF, Tanus-Santos JE. Effects of statins on matrix metalloproteinases and their endogenous inhibitors in human endothelial cells [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2011 ; 383( 6): 547-554.[citado 2026 mar. 15 ] Available from: https://doi.org/10.1007/s00210-011-0623-0 -
Vancouver
Izidoro-Toledo TC, Guimarães DA, Belo VA, Gerlach RF, Tanus-Santos JE. Effects of statins on matrix metalloproteinases and their endogenous inhibitors in human endothelial cells [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2011 ; 383( 6): 547-554.[citado 2026 mar. 15 ] Available from: https://doi.org/10.1007/s00210-011-0623-0 - Recombinant human matrix metalloproteinase-2 impairs cardiovascular 'beta'-adrenergic responses
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