Variants in S1P and ABCG1 genes are novel independent risk factors for dyslipidemia (2011)
- Authors:
- USP affiliated authors: DOREA, EGIDIO LIMA - HU ; BERNIK, MARCIA MARTINS SILVEIRA - HU ; HIRATA, MARIO HIROYUKI - FCF ; HIRATA, ROSARIO DOMINGUEZ CRESPO - FCF
- Unidades: HU; FCF
- Subjects: DISLIPIDEMIAS; HIPERCOLESTEROLEMIA; ARTERIOSCLEROSE
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Genética (SBG)
- Publisher place: Ribeirão Preto
- Date published: 2011
- Source:
- Conference titles: Congresso Brasileiro de Genética
-
ABNT
RODRIGUES, Alice Cristina et al. Variants in S1P and ABCG1 genes are novel independent risk factors for dyslipidemia. 2011, Anais.. Ribeirão Preto: Sociedade Brasileira de Genética (SBG), 2011. . Acesso em: 30 dez. 2025. -
APA
Rodrigues, A. C., Sobrino, B., Genvigir, F. D. V., Willrich, M. A. V., Arazi, S. S., Dórea, E. L., et al. (2011). Variants in S1P and ABCG1 genes are novel independent risk factors for dyslipidemia. In Resumos. Ribeirão Preto: Sociedade Brasileira de Genética (SBG). -
NLM
Rodrigues AC, Sobrino B, Genvigir FDV, Willrich MAV, Arazi SS, Dórea EL, Bernik MMS, Brion MJ, Carracedo A, Hirata MH, Hirata RDC. Variants in S1P and ABCG1 genes are novel independent risk factors for dyslipidemia. Resumos. 2011 ;[citado 2025 dez. 30 ] -
Vancouver
Rodrigues AC, Sobrino B, Genvigir FDV, Willrich MAV, Arazi SS, Dórea EL, Bernik MMS, Brion MJ, Carracedo A, Hirata MH, Hirata RDC. Variants in S1P and ABCG1 genes are novel independent risk factors for dyslipidemia. Resumos. 2011 ;[citado 2025 dez. 30 ] - CYP3A5 polymorphism is associated with differences on gene expression and cholesterol-lowering response to atorvastatin
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- Scavenger receptor class B type I polymorphisms and its relation with serum lipids profile and response to atorvastatin in Brazilian individuals
- mRNA expression of SREBF1, SREBF2 and SCAP transcription factors in peripheral mononuclear cells is modulated by atorvastatin therapy
- Resposta hipolipemiante à atorvastatina é modificada pelo polimorfismo do gene da CYP3A5
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