Microconversion between CYP21A2 and CYP21A1P Promoter Regions Causes the Nonclassical Form of 21-Hydroxylase Deficiency (2007)
- Authors:
- USP affiliated authors: MENDONÇA, BERENICE BILHARINHO DE - FM ; LIN, CHIN JIA - FM ; BACHEGA, TÂNIA APARECIDA SARTORI SANCHEZ - FM
- Unidade: FM
- DOI: 10.1210/jc.2006-2163
- Subjects: FENÓTIPOS (VARIAÇÃO); HIDROXILASE (DEFICIÊNCIA); DNA (ANÁLISE); GENOMAS
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2007
- Source:
- Título: Journal of Clinical Endocrinology and Metabolism
- ISSN: 0021-972X
- Volume/Número/Paginação/Ano: v. 92, n. 10, p. 4028-4034, 2007
- Status:
- Artigo possui acesso gratuito no site do editor (Bronze Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
ARAÚJO, Rogério S. et al. Microconversion between CYP21A2 and CYP21A1P Promoter Regions Causes the Nonclassical Form of 21-Hydroxylase Deficiency. Journal of Clinical Endocrinology and Metabolism, v. 92, n. 10, p. 4028-4034, 2007Tradução . . Disponível em: https://doi.org/10.1210/jc.2006-2163. Acesso em: 02 abr. 2026. -
APA
Araújo, R. S., Mendonça, B. B., Barbosa, Â. S., Lin, C. J., Marcondes, J. A. M., Billerbeck, A. E. C., & Bachega, T. A. S. S. (2007). Microconversion between CYP21A2 and CYP21A1P Promoter Regions Causes the Nonclassical Form of 21-Hydroxylase Deficiency. Journal of Clinical Endocrinology and Metabolism, 92( 10), 4028-4034. doi:10.1210/jc.2006-2163 -
NLM
Araújo RS, Mendonça BB, Barbosa ÂS, Lin CJ, Marcondes JAM, Billerbeck AEC, Bachega TASS. Microconversion between CYP21A2 and CYP21A1P Promoter Regions Causes the Nonclassical Form of 21-Hydroxylase Deficiency [Internet]. Journal of Clinical Endocrinology and Metabolism. 2007 ; 92( 10): 4028-4034.[citado 2026 abr. 02 ] Available from: https://doi.org/10.1210/jc.2006-2163 -
Vancouver
Araújo RS, Mendonça BB, Barbosa ÂS, Lin CJ, Marcondes JAM, Billerbeck AEC, Bachega TASS. Microconversion between CYP21A2 and CYP21A1P Promoter Regions Causes the Nonclassical Form of 21-Hydroxylase Deficiency [Internet]. Journal of Clinical Endocrinology and Metabolism. 2007 ; 92( 10): 4028-4034.[citado 2026 abr. 02 ] Available from: https://doi.org/10.1210/jc.2006-2163 - Avanços recentes no conhecimento dos mecanismos moleculares envolvidos na tumorigênese adrenocortical
- A possible role a radiation-induced p53 mutation in a poor outcome case of Nelson´s syndrome
- Plasma Renin Measurements are Unrelated to Mineralocorticoid Replacement Dose in Patients With Primary Adrenal Insufficiency
- CYP21A2 Genotypes do not Predict the Severity of Hyperandrogenic Manifestations in the Nonclassical Form of Congenital Adrenal Hyperplasia
- Long Term Glucocorticoid Exposition Does Not Play the Major Role in the Development of Obesity and Metabolic Syndrome in Classical Form of 21-Hydroxylase Deficiency
- The Presence of Clitoromegaly in the Nonclassical Form of 21-Hydroxylase Deficiency Could Be Partially Modulated by the CAG Polymorphic Tract of the Androgen Receptor Gene
- Long-term cardio-metabolic outcomes in patients with classical congenital adrenal hyperplasia: is the risk real?
- Impact of Glucocorticoid Receptor Gene Polymorphisms on the Metabolic Profile of Adult Patients with the Classical Form of 21-Hydroxylase Deficiency
- Clonality analysis and expression of ACTH and androgen receptors in giant myelolipomas
- Long-Term dexamethasone treatment is not associated with obesity and or metabolic syndrome in adult patients with classical forms of 21-hydroxylase deficiency
Informações sobre a disponibilidade de versões do artigo em acesso aberto coletadas automaticamente via oaDOI API (Unpaywall).
Por se tratar de integração com serviço externo, podem existir diferentes versões do trabalho (como preprints ou postprints), que podem diferir da versão publicada.
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas