Tracheal and aortic reactivity in a rat model of acute silicosis (2011)
- Authors:
- Autor USP: ROSSONI, LUCIANA VENTURINI - ICB
- Unidade: ICB
- Assunto: FISIOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Resumos
- Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental (FeSBE)
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ABNT
BUENO, F. R. et al. Tracheal and aortic reactivity in a rat model of acute silicosis. 2011, Anais.. São Paulo: FeSBE, 2011. . Acesso em: 25 mar. 2023. -
APA
Bueno, F. R., Machado, M. N., Zin, W. A., Rossoni, L. V., & Davel, A. P. C. (2011). Tracheal and aortic reactivity in a rat model of acute silicosis. In Resumos. São Paulo: FeSBE. -
NLM
Bueno FR, Machado MN, Zin WA, Rossoni LV, Davel APC. Tracheal and aortic reactivity in a rat model of acute silicosis. Resumos. 2011 ;[citado 2023 mar. 25 ] -
Vancouver
Bueno FR, Machado MN, Zin WA, Rossoni LV, Davel APC. Tracheal and aortic reactivity in a rat model of acute silicosis. Resumos. 2011 ;[citado 2023 mar. 25 ] - Effect of acute administration of 1 nM ouabaIN (OUA) on the vascular reactivity to pHenylephrine (phe) of the tail vascular bed from normotensive (wky) and hypertensive (shr) rats
- Nanomolar ouabain increases vascular reactivity to phenylephrine and functional activity of the Na, K-ATPase of tail vascular beds in normotensive (WKY) and hypertensive (SHR) rats
- Flow induces changes on the reactivity of the tail arterial bed from normotensive and hypertensive rats
- Effect of ageing and hypertension on the sodium pump activity in the rat aorta: Endothelial modulation
- Enhances in left ventricle function present in ouabain hypertensive rats are associated with changes in myosin ATPase activity and in expression of catalic isoforms of NA+, K+-ATPase
- Isoproterenol-induced cardiac hypertrophy is associated with endothelial dysfunction and vascular inflammation
- Fatores envolvidos na hiperatividade à noradrenalina em artérias de resistência de ratos hipertensos pelo tratamento por 20 semanas com oubaína
- Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation
- Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay
- Ajustes vasculares induzidos pela ouabaina na hipertensão
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