Haploinsufficiency for PKD1 results in increased tubularization of the bowman´s capsule and overexpression of COX-2 (2004)
- Authors:
- Autor USP: ONUCHIC, LUIZ FERNANDO - FM
- Unidade: FM
- Subjects: RIM (PATOLOGIA); CISTOS (GENÉTICA); MUTAÇÃO GENÉTICA; EXPERIMENTOS ANIMAIS; RATOS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of the American Society of Nephrology
- ISSN: 1046-6673
- Volume/Número/Paginação/Ano: v. 15, Abstracts issue, p. 219A, res. F-PO698, 2004
- Conference titles: Abstracts issue journal of the american Society of Nephrology - JASN
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ABNT
GONZALEZ, Miguel A. Garcia et al. Haploinsufficiency for PKD1 results in increased tubularization of the bowman´s capsule and overexpression of COX-2. Journal of the American Society of Nephrology. Baltimore: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 19 abr. 2024. , 2004 -
APA
Gonzalez, M. A. G., Piontek, K., Lijuan, L., Menezes, L. F., Onuchie, L., Lens, X. M., & Germino, G. (2004). Haploinsufficiency for PKD1 results in increased tubularization of the bowman´s capsule and overexpression of COX-2. Journal of the American Society of Nephrology. Baltimore: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Gonzalez MAG, Piontek K, Lijuan L, Menezes LF, Onuchie L, Lens XM, Germino G. Haploinsufficiency for PKD1 results in increased tubularization of the bowman´s capsule and overexpression of COX-2. Journal of the American Society of Nephrology. 2004 ; 15 219A.[citado 2024 abr. 19 ] -
Vancouver
Gonzalez MAG, Piontek K, Lijuan L, Menezes LF, Onuchie L, Lens XM, Germino G. Haploinsufficiency for PKD1 results in increased tubularization of the bowman´s capsule and overexpression of COX-2. Journal of the American Society of Nephrology. 2004 ; 15 219A.[citado 2024 abr. 19 ] - Regulação do volume celular no ramo ascendente fino da alça de Henle de ratos
- Fine mapping of the autosomal recessive polycystic kidney disease locus (PKHD1) and the genes MUT, RDS, CSNK2(beta), and GSTA1 at 6p21.1-p12
- Genomic structure and exclusion of two candidate genes for the PKHD1 locus and three new polymorphic markers within the critical interval
- Direct association of full-lenght PKD1 and PKD2 proteins in mammalian cells
- Aumento regulatório do volume celular em túbulos de malpighi
- Refinement of the autosomal recessive polycystic kidney disease (PKHD1) interval and exclusion of an EF hand-containing gene as a PKHD1 candidate gene
- Expression of PKD1 and PKD2 transcripts and proteins in human embryo and during normal kidney development
- Contribuições dos Modelos Animais ao Entendimento da Patogênese das Doenças Renais Policísticas
- Recombinant and endogenous polyductin (PD1) is secreted from primary cilia
- PKHD1 mutations in autosomal recessive polycystic kidney disease (ARPKD)
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