Redox regulation of the mitochondrial 'K IND. ATP' channel in cardioprotection (2011)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1016/j.bbamcr.2010.11.005
- Subjects: ÁCIDOS GRAXOS; ÓXIDO NÍTRICO; RADICAIS LIVRES
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Biochimica et Biophysica Acta - Molecular Cell Research
- ISSN: 0167-4889
- Volume/Número/Paginação/Ano: v. 1813, n. 7, p. 1309-1315 : + Supplementary materials (p. S1-S8), 2011
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: publisher-specific-oa
-
ABNT
QUELICONI, Bruno Barros et al. Redox regulation of the mitochondrial 'K IND. ATP' channel in cardioprotection. Biochimica et Biophysica Acta - Molecular Cell Research, v. 1813, n. 7, p. 1309-1315 : + Supplementary materials ( S1-S8), 2011Tradução . . Disponível em: https://doi.org/10.1016/j.bbamcr.2010.11.005. Acesso em: 19 abr. 2024. -
APA
Queliconi, B. B., Wojtovich, A. P., Nadtochiy, S. M., Kowaltowski, A. J., & Brookes, P. S. (2011). Redox regulation of the mitochondrial 'K IND. ATP' channel in cardioprotection. Biochimica et Biophysica Acta - Molecular Cell Research, 1813( 7), 1309-1315 : + Supplementary materials ( S1-S8). doi:10.1016/j.bbamcr.2010.11.005 -
NLM
Queliconi BB, Wojtovich AP, Nadtochiy SM, Kowaltowski AJ, Brookes PS. Redox regulation of the mitochondrial 'K IND. ATP' channel in cardioprotection [Internet]. Biochimica et Biophysica Acta - Molecular Cell Research. 2011 ; 1813( 7): 1309-1315 : + Supplementary materials ( S1-S8).[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.bbamcr.2010.11.005 -
Vancouver
Queliconi BB, Wojtovich AP, Nadtochiy SM, Kowaltowski AJ, Brookes PS. Redox regulation of the mitochondrial 'K IND. ATP' channel in cardioprotection [Internet]. Biochimica et Biophysica Acta - Molecular Cell Research. 2011 ; 1813( 7): 1309-1315 : + Supplementary materials ( S1-S8).[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.bbamcr.2010.11.005 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1016/j.bbamcr.2010.11.005 (Fonte: oaDOI API)
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