Dose-response met-rantes treatment of experimental periodontitis: a narrow edge between the disease severity attenuation and infection control (2011)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; SANTOS, CARLOS FERREIRA DOS - FOB ; CAMPANELLI, ANA PAULA - FOB ; GARLET, GUSTAVO POMPERMAIER - FOB ; CAMPOS, MARIO JULIO AVILA - ICB
- Unidades: FMRP; FOB; ICB
- DOI: 10.1371/journal.pone.0022526
- Subjects: PERIODONTITE (TRATAMENTO); RECEPTORES IMUNOLÓGICOS; CONTROLE DE INFECÇÕES
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2011
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
REPEKE, Carlos Eduardo et al. Dose-response met-rantes treatment of experimental periodontitis: a narrow edge between the disease severity attenuation and infection control. PLOS ONE, v. 6, n. 7, p. e22526, 2011Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0022526. Acesso em: 19 abr. 2024. -
APA
Repeke, C. E., Ferreira Jr., S. B., Vieira, A. E., Silveira, E. M., Avila-Campos, M. J., Silva, J. S. da, et al. (2011). Dose-response met-rantes treatment of experimental periodontitis: a narrow edge between the disease severity attenuation and infection control. PLOS ONE, 6( 7), e22526. doi:10.1371/journal.pone.0022526 -
NLM
Repeke CE, Ferreira Jr. SB, Vieira AE, Silveira EM, Avila-Campos MJ, Silva JS da, Santos CF dos, Campanelli AP, Trombone APF, Garlet GP. Dose-response met-rantes treatment of experimental periodontitis: a narrow edge between the disease severity attenuation and infection control [Internet]. PLOS ONE. 2011 ; 6( 7): e22526.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1371/journal.pone.0022526 -
Vancouver
Repeke CE, Ferreira Jr. SB, Vieira AE, Silveira EM, Avila-Campos MJ, Silva JS da, Santos CF dos, Campanelli AP, Trombone APF, Garlet GP. Dose-response met-rantes treatment of experimental periodontitis: a narrow edge between the disease severity attenuation and infection control [Internet]. PLOS ONE. 2011 ; 6( 7): e22526.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1371/journal.pone.0022526 - An interleukin-1beta (IL-1ß) single-nucleotide polymorphism at position 3954 and red complex periodontopathogens independently and additively modulate the levels of IL-1ß in diseased periodontal tissues
- Functional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in mice
- TNF-alpha/CCL3-5/CCR5 cascade mediates RANKL plus cells migration in inflammatory bone resorption
- Association of human T lymphotropic virus 1 amplification of periodontitis severity with altered cytokine expression in response to a standard periodontopathogen infection
- Toll-like receptor 4 is expressed in pulp fibroblasts after stimulation with Candida Albicans
- Recognition of Candida albicans by gingival fibroblasts: the role of TLR2, TLR4/CD14, and MyD88
- Tumor necrosis factor-alpha -308G/A single nucleotide polymorphism and red-complex periodontopathogens are independently associated with increased levels of tumor necrosis factor-alpha in diseased periodontal tissues
- Experimental periodontitis in mice selected for maximal or minimal inflammatory reactions: increased inflammatory immune responsiveness drives increased alveolar bone loss without enhancing the control of periodontal infection
- The role of toll-Like receptor 2 in the recognition of Aggregatibacter actinomycetemcomitans
- CD25+ T cell depletion impairs murine squamous cell carcinoma development via modulation of antitumor immune responses
Informações sobre o DOI: 10.1371/journal.pone.0022526 (Fonte: oaDOI API)
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