Regulation of Trypanosoma cruzi-induced myocarditis by programmed death cell receptor 1 (2011)
- Authors:
- USP affiliated authors: CAMPANELLI, ANA PAULA - FOB ; SILVA, JOÃO SANTANA DA - FMRP
- Unidades: FOB; FMRP
- DOI: 10.1128/IAI.01047-10
- Subjects: TRYPANOSOMA CRUZI; MIOCARDITE; DOENÇA DE CHAGAS
- Language: Inglês
- Imprenta:
- Source:
- Título: Infection and Immunity
- ISSN: 0019-9567
- Volume/Número/Paginação/Ano: v. 79, n. 5, p. 1873-1881, May 2011
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
GUTIERREZ, Fredy R. S. et al. Regulation of Trypanosoma cruzi-induced myocarditis by programmed death cell receptor 1. Infection and Immunity, v. 79, n. 5, p. 1873-1881, 2011Tradução . . Disponível em: https://doi.org/10.1128/IAI.01047-10. Acesso em: 13 abr. 2026. -
APA
Gutierrez, F. R. S., Mariano, F. S., Oliveira, C. J. F., Pavanelli, W. R., Guedes, P. M. M., Silva, G. K., et al. (2011). Regulation of Trypanosoma cruzi-induced myocarditis by programmed death cell receptor 1. Infection and Immunity, 79( 5), 1873-1881. doi:10.1128/IAI.01047-10 -
NLM
Gutierrez FRS, Mariano FS, Oliveira CJF, Pavanelli WR, Guedes PMM, Silva GK, Campanelli AP, Milanezi CM, Azuma M, Honjo T, Teixeira MM, Aliberti JCS, Silva JS da. Regulation of Trypanosoma cruzi-induced myocarditis by programmed death cell receptor 1 [Internet]. Infection and Immunity. 2011 ; 79( 5): 1873-1881.[citado 2026 abr. 13 ] Available from: https://doi.org/10.1128/IAI.01047-10 -
Vancouver
Gutierrez FRS, Mariano FS, Oliveira CJF, Pavanelli WR, Guedes PMM, Silva GK, Campanelli AP, Milanezi CM, Azuma M, Honjo T, Teixeira MM, Aliberti JCS, Silva JS da. Regulation of Trypanosoma cruzi-induced myocarditis by programmed death cell receptor 1 [Internet]. Infection and Immunity. 2011 ; 79( 5): 1873-1881.[citado 2026 abr. 13 ] Available from: https://doi.org/10.1128/IAI.01047-10 - Expression of suppressors of cytokine signaling in diseased periodontal tissues: a stop signal for disease progression?
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