The pattern recognition receptors Nod1 and Nod2 account for neutrophil recruitment to the lungs of mice infected with Legionella pneumophila (2010)
- Authors:
- USP affiliated authors: CUNHA, FERNANDO DE QUEIROZ - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.micinf.2010.05.006
- Subjects: LEGIONELLA PNEUMOPHILA; NEUTRÓFILOS
- Language: Inglês
- Imprenta:
- Publisher place: Les Moulineaux
- Date published: 2010
- Source:
- Título: Microbes and Infection
- ISSN: 1986-4579
- Volume/Número/Paginação/Ano: v. 12, n. 11, p. 819-827, 2010
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
FRUTUOSO, Mariana Shumaher et al. The pattern recognition receptors Nod1 and Nod2 account for neutrophil recruitment to the lungs of mice infected with Legionella pneumophila. Microbes and Infection, v. 12, n. 11, p. 819-827, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2010.05.006. Acesso em: 11 fev. 2026. -
APA
Frutuoso, M. S., Hori, J. I., Pereira, M. S. F., Lima Júnior, D. de S., Sônego, F., Kobayashi, K. S., et al. (2010). The pattern recognition receptors Nod1 and Nod2 account for neutrophil recruitment to the lungs of mice infected with Legionella pneumophila. Microbes and Infection, 12( 11), 819-827. doi:10.1016/j.micinf.2010.05.006 -
NLM
Frutuoso MS, Hori JI, Pereira MSF, Lima Júnior D de S, Sônego F, Kobayashi KS, Flavell RA, Cunha F de Q, Zamboni DS. The pattern recognition receptors Nod1 and Nod2 account for neutrophil recruitment to the lungs of mice infected with Legionella pneumophila [Internet]. Microbes and Infection. 2010 ; 12( 11): 819-827.[citado 2026 fev. 11 ] Available from: https://doi.org/10.1016/j.micinf.2010.05.006 -
Vancouver
Frutuoso MS, Hori JI, Pereira MSF, Lima Júnior D de S, Sônego F, Kobayashi KS, Flavell RA, Cunha F de Q, Zamboni DS. The pattern recognition receptors Nod1 and Nod2 account for neutrophil recruitment to the lungs of mice infected with Legionella pneumophila [Internet]. Microbes and Infection. 2010 ; 12( 11): 819-827.[citado 2026 fev. 11 ] Available from: https://doi.org/10.1016/j.micinf.2010.05.006 - Mechanism of virulence in the murine moldel of legionella longbeachae infection
- Unraveling the pathogenesis of Legionella longbeachae infection: a role of type IV secretion system to induce mice lethality via pulmonary inflammation
- The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88
- Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain
- The role of NLRP12 in periapical lesion progression
- Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis
- NOD1 and NOD2 contribute to pain hypersensitivity and glial cells activation after induction of peripheral nerve injury
- NLRP3 inflammasome–mediated neutrophil recruitment and hypernociception depend on leukotriene B4 in a murine model of gout
- MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response
- Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection
Informações sobre o DOI: 10.1016/j.micinf.2010.05.006 (Fonte: oaDOI API)
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