Involvement of cAMP/Epac/PI3K-dependent pathway in the antiproteolytic effect epinephrine on rat skeletal muscle (2010)
- Authors:
- USP affiliated authors: ZANON, NEUSA MARIA - FMRP ; NAVEGANTES, LUIZ CARLOS CARVALHO - FMRP ; KETTELHUT, ISIS DO CARMO - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.mce.2009.09.028
- Subjects: MÚSCULO ESQUELÉTICO; METABOLISMO DE PROTEÍNA
- Language: Inglês
- Imprenta:
- Source:
- Título: Molecular and Cellular Endocrinology
- ISSN: 0303-7207
- Volume/Número/Paginação/Ano: v. 315, n. 1/2, p. 104-112, 2010
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: unspecified-oa
-
ABNT
BAVIERA, Amanda Martins et al. Involvement of cAMP/Epac/PI3K-dependent pathway in the antiproteolytic effect epinephrine on rat skeletal muscle. Molecular and Cellular Endocrinology, v. 315, n. 1/2, p. 104-112, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2009.09.028. Acesso em: 09 out. 2024. -
APA
Baviera, A. M., Zanon, N. M., Navegantes, L. C. C., & Kettelhut, I. do C. (2010). Involvement of cAMP/Epac/PI3K-dependent pathway in the antiproteolytic effect epinephrine on rat skeletal muscle. Molecular and Cellular Endocrinology, 315( 1/2), 104-112. doi:10.1016/j.mce.2009.09.028 -
NLM
Baviera AM, Zanon NM, Navegantes LCC, Kettelhut I do C. Involvement of cAMP/Epac/PI3K-dependent pathway in the antiproteolytic effect epinephrine on rat skeletal muscle [Internet]. Molecular and Cellular Endocrinology. 2010 ; 315( 1/2): 104-112.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.mce.2009.09.028 -
Vancouver
Baviera AM, Zanon NM, Navegantes LCC, Kettelhut I do C. Involvement of cAMP/Epac/PI3K-dependent pathway in the antiproteolytic effect epinephrine on rat skeletal muscle [Internet]. Molecular and Cellular Endocrinology. 2010 ; 315( 1/2): 104-112.[citado 2024 out. 09 ] Available from: https://doi.org/10.1016/j.mce.2009.09.028 - Efeito da peçonha de bothrops Jararaca no metabolismo protéico em músculos esqueléticos de ratos
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- Catecholamines control the lysosomal proteolytic process in rat cardiac muscle
- Insulin deficiency induces heart atrophy by activation of lysosomal and ubiquitin-proteasome proteolytic pathways in rats
- 'beta IND.2'-adrenergic agonists reactivate ERK1/2 and Akt signaling and supress atrophic genes in skeletal muscles from fasted mice
- Acute cold exposure increases proteolysis but does not change the rates of protein synthesis in skeletal muscles of rats
- Differences in antiproteolytic response to ''beta'IND.2'-adrenergic agonist in white and red skeletal muscles
- Norepinephrine attenuates the fasting-induced expression of atrophy- and autophagy-related genes in skeletal muscles of mice
- Insulin suppresses atrophy- and autophagy-related genes in heart tissue and cardiomyocytes through AKT/FOXO signaling
Informações sobre o DOI: 10.1016/j.mce.2009.09.028 (Fonte: oaDOI API)
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