Changes in glomerular capillary walls from rats exposed to losartan during lactation (2009)
- Authors:
- USP affiliated authors: COSTA, ROBERTO SILVA - FMRP ; SILVA, CLEONICE GIOVANINI ALVES DA - FMRP ; COIMBRA, TEREZILA MACHADO - FMRP
- Unidade: FMRP
- Subjects: NEFROPATIAS; ANGIOTENSINA II
- Language: Inglês
- Imprenta:
- Publisher: American Society of Nephrology
- Publisher place: San Diego
- Date published: 2009
- Source:
- Título: Preliminary Program
- Conference titles: Renal Week
-
ABNT
MARIN, Evelyn Cristina Santana et al. Changes in glomerular capillary walls from rats exposed to losartan during lactation. 2009, Anais.. San Diego: American Society of Nephrology, 2009. . Acesso em: 11 jan. 2026. -
APA
Marin, E. C. S., Costa, R. S., Silva, C. G. A. da, & Coimbra, T. M. (2009). Changes in glomerular capillary walls from rats exposed to losartan during lactation. In Preliminary Program. San Diego: American Society of Nephrology. -
NLM
Marin ECS, Costa RS, Silva CGA da, Coimbra TM. Changes in glomerular capillary walls from rats exposed to losartan during lactation. Preliminary Program. 2009 ;[citado 2026 jan. 11 ] -
Vancouver
Marin ECS, Costa RS, Silva CGA da, Coimbra TM. Changes in glomerular capillary walls from rats exposed to losartan during lactation. Preliminary Program. 2009 ;[citado 2026 jan. 11 ] - Postnatal renal abnormalities in rats exposed to losartan during lactation
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- Early events in rats exposed to losartan during lactation
- The oxidative stress role in renal injury induced in rats by losartan exposition during lactation
- Inhibition of endogenous hydrogen sulfide formation reduces renal damage induced by adriamycin injection
- Inhibition of endogenous hydrogen sulfide formation reduces renal damage induced by cisplatin injection
- Resveratrol attenuates Glycerol-induced renal injury
- Participação da albumina nas lesões túbulo-intersticiais induzidas pela adriamicina (ADRI)
- Angiotensin II and endothelin in the renal cortex during the evolution of glycerol-induced acute tubular necrosis
- O aumento da ingestão de sodio reduz as alterações renais e a excreção urinária de TG-F-'BETA' ratas tratadas com adriamicina
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