Regulatory T cells attenuate experimental periodontitis progression in mice (2010)
- Authors:
- USP affiliated authors: ASSIS, GERSON FRANCISCO DE - FOB ; CAMPANELLI, ANA PAULA - FOB ; GARLET, GUSTAVO POMPERMAIER - FOB ; SILVA, JOÃO SANTANA DA - FMRP ; CAMPOS, MARIO JULIO AVILA - ICB
- Unidades: FOB; FMRP; ICB
- DOI: 10.1111/j.1600-051X.2010.01586.x
- Subjects: CITOCINAS; DOENÇAS PERIODONTAIS (IMUNOLOGIA); LINFÓCITOS T; MICROBIOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Copenhagen
- Date published: 2010
- Source:
- Título do periódico: Journal of Clinical Periodontology
- ISSN: 0303-6979
- Volume/Número/Paginação/Ano: v. 37, n. 7, p. 591-600, 2010
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
GARLET, Gustavo Pompermaier et al. Regulatory T cells attenuate experimental periodontitis progression in mice. Journal of Clinical Periodontology, v. 37, n. 7, p. 591-600, 2010Tradução . . Disponível em: https://doi.org/10.1111/j.1600-051X.2010.01586.x. Acesso em: 29 set. 2024. -
APA
Garlet, G. P., Cardoso, C. R., Mariano, F. S., Claudino, M., Assis, G. F. de, Campanelli, A. P., et al. (2010). Regulatory T cells attenuate experimental periodontitis progression in mice. Journal of Clinical Periodontology, 37( 7), 591-600. doi:10.1111/j.1600-051X.2010.01586.x -
NLM
Garlet GP, Cardoso CR, Mariano FS, Claudino M, Assis GF de, Campanelli AP, Ávila-Campos MJ, Silva JS da. Regulatory T cells attenuate experimental periodontitis progression in mice [Internet]. Journal of Clinical Periodontology. 2010 ; 37( 7): 591-600.[citado 2024 set. 29 ] Available from: https://doi.org/10.1111/j.1600-051X.2010.01586.x -
Vancouver
Garlet GP, Cardoso CR, Mariano FS, Claudino M, Assis GF de, Campanelli AP, Ávila-Campos MJ, Silva JS da. Regulatory T cells attenuate experimental periodontitis progression in mice [Internet]. Journal of Clinical Periodontology. 2010 ; 37( 7): 591-600.[citado 2024 set. 29 ] Available from: https://doi.org/10.1111/j.1600-051X.2010.01586.x - Periodontitis and arthritis interaction in mice involves a shared hyper-inflammatory genotype and functional immunological interferences
- Evidences of the cooperative role of the chemokines CCL3, CCL4 and CCL5 and its receptors CCR1+ and CCR5+ in RANKL+ cell migration throughout experimental periodontitis in mice
- TNF-alpha/CCL3-5/CCR5 cascade mediates RANKL plus cells migration in inflammatory bone resorption
- Association of human T lymphotropic virus 1 amplification of periodontitis severity with altered cytokine expression in response to a standard periodontopathogen infection
- Functional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in mice
- The cooperative role of CCR1 and CCR5 in the migration of osteoclast precursors and RANKL+ Cells throughout inflammatory bone loss after periodontal infection
- Experimental periodontitis in mice selected for maximal or minimal inflammatory reactions: increased inflammatory immune responsiveness drives increased alveolar bone loss without enhancing the control of periodontal infection
- Interaction between experimental periodontitis and arthritis involves shared genetic susceptibility and functional interferences
- The role of MIP-1A (CCL3) on the immunomodulation of experimental periodontal disease in mice
- Shared genetic susceptibility and functional interferences in the interaction between periodontitis and arthritis: a possible role for NRAMP1/SLC11A1
Informações sobre o DOI: 10.1111/j.1600-051X.2010.01586.x (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas