Differential expression of HNF4α and genes related to genesis, metabolism and growth in culture neonatal pancreatic islets (2008)
- Authors:
- USP affiliated authors: SILVA, SILVANA AUXILIADORA BORDIN DA - ICB
- Unidades: ICB
- Subjects: FISIOLOGIA
- Language: Inglês
- Imprenta:
- Conference title: Symposium Mechanisms of Insulin Secretion: molecular and functional aspects
-
ABNT
LEITE, A. R.; SILVA, Silvana Auxiliadora Bordin da. Differential expression of HNF4α and genes related to genesis, metabolism and growth in culture neonatal pancreatic islets. Anais.. São Paulo: [s.n.], 2008. -
APA
Leite, A. R., & Silva, S. A. B. da. (2008). Differential expression of HNF4α and genes related to genesis, metabolism and growth in culture neonatal pancreatic islets. In . São Paulo. -
NLM
Leite AR, Silva SAB da. Differential expression of HNF4α and genes related to genesis, metabolism and growth in culture neonatal pancreatic islets. 2008 ; -
Vancouver
Leite AR, Silva SAB da. Differential expression of HNF4α and genes related to genesis, metabolism and growth in culture neonatal pancreatic islets. 2008 ; - Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway
- A low protein diet alters gene expression in rat pancreatic islets
- Participação do STAT3 na regressão do pâncreas endócrino no período perinatal:: papel da prolactina e dos glicocorticóides
- Alterações funcionais da ilhota pancreática durante a gestação e lactação
- Tratamento ICV agudo com TNFa aumenta secreção de insulina e leva a alterações na expressão gênica em ilhotas
- Islet Neogenesis Associated Protein activates several proteins involved in growth and differentiation of neonatal rat pancreatic islets: possible involvement of cholinergic pathway
- Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus
- BMP-9 modula agudamente a fosforilação da AKT induzida por insulina em músculo oxidativo de ratos
- UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression
- Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats
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