Angiotensin II vascular Hyperreactivity in contralateral artery after ballon catheter injury: ROS and nitric oxide pathway modulation (2009)
- Authors:
- USP affiliated authors: RAMALHO, LEANDRA NAIRA ZAMBELLI - FMRP ; OLIVEIRA, ANA MARIA DE - FCFRP
- Unidades: FMRP; FCFRP
- Subjects: ANGIOTENSINA II; ANGIOPLASTIA COM BALÃO; ARTÉRIAS CARÓTIDAS; ÓXIDO NÍTRICO
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 2009
- Source:
- Título do periódico: Abstracts
- Conference titles: Annual Conference of The American Heart Association
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ABNT
OLIVON, Vania C. e RAMALHO, Leandra Naira Zambelli e OLIVEIRA, Ana Maria de. Angiotensin II vascular Hyperreactivity in contralateral artery after ballon catheter injury: ROS and nitric oxide pathway modulation. 2009, Anais.. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2009. . Acesso em: 24 abr. 2024. -
APA
Olivon, V. C., Ramalho, L. N. Z., & Oliveira, A. M. de. (2009). Angiotensin II vascular Hyperreactivity in contralateral artery after ballon catheter injury: ROS and nitric oxide pathway modulation. In Abstracts. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Olivon VC, Ramalho LNZ, Oliveira AM de. Angiotensin II vascular Hyperreactivity in contralateral artery after ballon catheter injury: ROS and nitric oxide pathway modulation. Abstracts. 2009 ;[citado 2024 abr. 24 ] -
Vancouver
Olivon VC, Ramalho LNZ, Oliveira AM de. Angiotensin II vascular Hyperreactivity in contralateral artery after ballon catheter injury: ROS and nitric oxide pathway modulation. Abstracts. 2009 ;[citado 2024 abr. 24 ] - The balloon catheter induces an increased to angiotensin II in reactivity on distant from the injury site
- The participation of neurocompensatory mechanisms in the increased ANG-II-Induced contraction in contralateral carotid after balloon catheter
- Cellular mechanisms underlying increased phe-induced contraction in contralateral carotid after balloon catheter
- Reactive oxygen species participation on angiotensin II-induced vascular hyper-reactivity in contralateral artery after balloon catheter injury
- Consequences of acute or chronic stress on relaxation induced by angiotensin 1-7 in rat carotid
- Vascular hyper-reactivity following arterial balloon injury: ros and nitric oxide participation on distant and dalayed effects
- The role of endothelium in the increased in vascular hyper-reactivity following arterial ballon injury
- Perivascular injury leads to a reduction in vascular reactivity of the collared and to an enhancement on contralateral carotid artery of rats
- Vascular hyper-reactivity following arterial ballon injury: ROS and prostanoids participation on distant and dalayed effects
- The role of nitric oxide pathway in the increased angiotensin II-induced contraction in contralateral artery after balloon catheter injury
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