Chronic intermittent hypoxia alters the respiratory and autonomic responses to activation of glutamate receptors in the caudal NTS of juvenile rats (2009)
- Authors:
- Autor USP: MACHADO, BENEDITO HONÓRIO - FMRP
- Unidade: FMRP
- Subjects: ANÓXIA; RECEPTORES DE GLUTAMATO; RATOS (FISIOLOGIA)
- Language: Inglês
- Imprenta:
- Publisher place: New Orleans
- Date published: 2009
- Source:
- Título: Abstracts
- Conference titles: Experimental Biology
-
ABNT
ZOCCAL, Daniel B. e MACHADO, Benedito Honório. Chronic intermittent hypoxia alters the respiratory and autonomic responses to activation of glutamate receptors in the caudal NTS of juvenile rats. 2009, Anais.. New Orleans: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2009. . Acesso em: 17 jan. 2026. -
APA
Zoccal, D. B., & Machado, B. H. (2009). Chronic intermittent hypoxia alters the respiratory and autonomic responses to activation of glutamate receptors in the caudal NTS of juvenile rats. In Abstracts. New Orleans: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Zoccal DB, Machado BH. Chronic intermittent hypoxia alters the respiratory and autonomic responses to activation of glutamate receptors in the caudal NTS of juvenile rats. Abstracts. 2009 ;[citado 2026 jan. 17 ] -
Vancouver
Zoccal DB, Machado BH. Chronic intermittent hypoxia alters the respiratory and autonomic responses to activation of glutamate receptors in the caudal NTS of juvenile rats. Abstracts. 2009 ;[citado 2026 jan. 17 ] - Evidências de que as alterações no acoplamento simpático-respiratório após a expulsão à hipóxia crônica intermitente são independentes de células gliais no NTS
- Acute inhibition of glial cells in the NTS does not affect respiratory and sympathetic activities in rats exposed to chronic intermittent hypoxia
- Glutamatergic mechanisms on the interaction of sympathetic and respiratory responses to chemoreflex activation in the NTS
- Neural mechanisms of cardiovascular regulation
- Effect of nucleus ambiguus lesion on the development of neurogenic hypertension
- Antagonism of glutamatergic ionotropic receptors in the iNTS and cNTS of rats alters the basal respiratory rhythm and attenuates the peripheral chemoreflex responses
- P2 receptors activation increases L-glutamate release in NTS neurons projecting to RVLM in rat brainstem slices
- Neuro transmission of the chemoreflex sympatho-excitation in the nucleus tractus solitarii of rats in different experimental models
- The inhibition of GAP junctions in the caudal NTS (cNTS) alters the baseline phrenic nerve discharge and sympathetic activity
- Autonomic and respiratory responses to microinjection of L-glutamate into the commissural subnucleus of the NTS in the working heart-brainstem preparation of the rat
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