Modulatory activity of flavonoids in the human neutrophil oxidative metabolism and phagocytosis stimulated via FC-gamma and complement receptors (2008)
- Authors:
- USP affiliated authors: VALIM, YARA MARIA LUCISANO - FCFRP ; BARUFFI, MARCELO DIAS - FCFRP ; AZZOLINI, ANA ELISA CALEIRO SEIXAS - FCFRP
- School: FCFRP
- Subjects: NEUTRÓFILOS (HUMANO); FLAVONÓIDES
- Language: Inglês
- Imprenta:
- Place of publication: Ribeirão Preto
- Date published: 2008
- Source:
- Título do periódico: Abstracts
- Conference title: Meeting of the Brazilian Society for Immunology
-
ABNT
SANTOS, Everton O. L.; AZZOLINI, Ana Elisa Caleiro Seixas; SANTOS, Marilia F. dos; et al. Modulatory activity of flavonoids in the human neutrophil oxidative metabolism and phagocytosis stimulated via FC-gamma and complement receptors. Anais.. Ribeirão Preto: [s.n.], 2008. -
APA
Santos, E. O. L., Azzolini, A. E. C. S., Santos, M. F. dos, Pancoto, J. A. T., Baruffi, M. D., & Lucisano-Valim, Y. M. (2008). Modulatory activity of flavonoids in the human neutrophil oxidative metabolism and phagocytosis stimulated via FC-gamma and complement receptors. In Abstracts. Ribeirão Preto. -
NLM
Santos EOL, Azzolini AECS, Santos MF dos, Pancoto JAT, Baruffi MD, Lucisano-Valim YM. Modulatory activity of flavonoids in the human neutrophil oxidative metabolism and phagocytosis stimulated via FC-gamma and complement receptors. Abstracts. 2008 ; -
Vancouver
Santos EOL, Azzolini AECS, Santos MF dos, Pancoto JAT, Baruffi MD, Lucisano-Valim YM. Modulatory activity of flavonoids in the human neutrophil oxidative metabolism and phagocytosis stimulated via FC-gamma and complement receptors. Abstracts. 2008 ; - Galectin-1 modulation of neutrophil reactive oxygen species production depends on the cell activation state
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- Microbial killing and reactive oxygen species generation by human neitrophils triggered by different signaling pathways: modulatory effects of four flavonols
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- Interleukin-8-induced priming of circulating neutrophils from patients with rheumatoid arthritis: increase of oxidative burst and decrease of apoptosis
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