Chronic paroxetine and alprazolam decrease the hypothalamic activity in rodents threatened by venomous coral snake (2008)
- Authors:
- Autor USP: COIMBRA, NORBERTO CYSNE - FMRP
- Unidade: FMRP
- Subjects: NEUROANATOMIA; FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: European Neuropsychopharmacology
- ISSN: 0924-977X
- Volume/Número/Paginação/Ano: v. 18, suppl. 4, p. S222, res. P.1.c.003, 2008
- Conference titles: European College of Neuropsychopharmacology of Congress (ECNP)
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ABNT
PASCHOALIM-MAURIN, T. e COIMBRA, Norberto Cysne. Chronic paroxetine and alprazolam decrease the hypothalamic activity in rodents threatened by venomous coral snake. European Neuropsychopharmacology. Amsterdam: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 23 jan. 2026. , 2008 -
APA
Paschoalim-Maurin, T., & Coimbra, N. C. (2008). Chronic paroxetine and alprazolam decrease the hypothalamic activity in rodents threatened by venomous coral snake. European Neuropsychopharmacology. Amsterdam: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Paschoalim-Maurin T, Coimbra NC. Chronic paroxetine and alprazolam decrease the hypothalamic activity in rodents threatened by venomous coral snake. European Neuropsychopharmacology. 2008 ; 18 S222.[citado 2026 jan. 23 ] -
Vancouver
Paschoalim-Maurin T, Coimbra NC. Chronic paroxetine and alprazolam decrease the hypothalamic activity in rodents threatened by venomous coral snake. European Neuropsychopharmacology. 2008 ; 18 S222.[citado 2026 jan. 23 ] - Opioid neurotransmission modulates defensive behavior and fear-induced antinociception in dangerous environments
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- Naloxone-induced antipanic effect on defensive behaviour and innate fear-induced antinociception of preys confronted with rattlesnakes
- Chronic paroxetine and alprazolam use PAG columns as pharmacological targets
- Estudo das conexoes nigro-tectais com tracadores fluorescentes
- Nicotinic and muscarinic cholinergic receptors are recruited by acetylcholine-mediated neurotransmission within the locus coeruleus during the organisation of post-ictal antinociception
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