Association of mutant SOD to DNA may be related to increased DNA damage, p53 activation and apoptosis in a cellular model of amyotrophic lateral sclerosis (2007)
- Authors:
- USP affiliated authors: SOGAYAR, MARI CLEIDE - IQ ; AUGUSTO, OHARA - IQ ; MASCIO, PAOLO DI - IQ ; MEDEIROS, MARISA HELENA GENNARI DE - IQ
- Unidade: IQ
- Subjects: DANO AO DNA; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher: Facultad de Medicina/Departamento de Bioquímica
- Publisher place: Montevideo
- Date published: 2007
- Source:
- Título do periódico: Abstracts
- Conference titles: Free Radicals in Montevideo
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ABNT
BARBOSA, Lívea Fujita et al. Association of mutant SOD to DNA may be related to increased DNA damage, p53 activation and apoptosis in a cellular model of amyotrophic lateral sclerosis. 2007, Anais.. Montevideo: Facultad de Medicina/Departamento de Bioquímica, 2007. . Acesso em: 24 abr. 2024. -
APA
Barbosa, L. F., Cerqueira, F. M., Macedo, A. F. de A., Sogayar, M. C., Augusto, O., Carri, M. T., et al. (2007). Association of mutant SOD to DNA may be related to increased DNA damage, p53 activation and apoptosis in a cellular model of amyotrophic lateral sclerosis. In Abstracts. Montevideo: Facultad de Medicina/Departamento de Bioquímica. -
NLM
Barbosa LF, Cerqueira FM, Macedo AF de A, Sogayar MC, Augusto O, Carri MT, Di Mascio P, Medeiros MHG de. Association of mutant SOD to DNA may be related to increased DNA damage, p53 activation and apoptosis in a cellular model of amyotrophic lateral sclerosis. Abstracts. 2007 ;[citado 2024 abr. 24 ] -
Vancouver
Barbosa LF, Cerqueira FM, Macedo AF de A, Sogayar MC, Augusto O, Carri MT, Di Mascio P, Medeiros MHG de. Association of mutant SOD to DNA may be related to increased DNA damage, p53 activation and apoptosis in a cellular model of amyotrophic lateral sclerosis. Abstracts. 2007 ;[citado 2024 abr. 24 ] - DNA damage, p53 activation and increased apoptosis in human neuroblastoma cells expressing a mutant SOD typical of familial amyotrophic lateral sclerosis (FALS)
- DNA damage, P53 activation and apoptosis in a cellular model of amyotrophic lateral sclerosis
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