Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria (2007)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: MITOCÔNDRIAS; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher: Facultad de Medicina/Departamento de Bioquímica
- Publisher place: Montevideo
- Date published: 2007
- Source:
- Título: Abstracts
- Conference titles: Free Radicals in Montevideo
-
ABNT
FORNAZARI, Maynara et al. Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria. 2007, Anais.. Montevideo: Facultad de Medicina/Departamento de Bioquímica, 2007. . Acesso em: 20 jan. 2026. -
APA
Fornazari, M., Paula, J. G. de, Castilho, R. F., & Kowaltowski, A. J. (2007). Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria. In Abstracts. Montevideo: Facultad de Medicina/Departamento de Bioquímica. -
NLM
Fornazari M, Paula JG de, Castilho RF, Kowaltowski AJ. Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria. Abstracts. 2007 ;[citado 2026 jan. 20 ] -
Vancouver
Fornazari M, Paula JG de, Castilho RF, Kowaltowski AJ. Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria. Abstracts. 2007 ;[citado 2026 jan. 20 ] - Higher respiratory activity decreases mitochondrial reactive oxygen release and increases life span in Saccharomyces cerevisiae
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- Respiration, oxidative stress and chronological life span in S. cerevisiae
- ATP-sensitive 'K POT +'channels in brain mitochondria are redox-sensitive and regulate reactive oxygen release
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Phosphate uptake increases mitochondrial H2O2 release
- How mitochondrial complex II inhibition promotes neuronal damage
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