Variants in the promoter region of IKBL/NFKBIL1 gene may mark susceptibility to the development of chronic Chagas' cardiomyopathy among Trypanosoma cruzi-infected individuals (2008)
- Authors:
- USP affiliated authors: CUNHA NETO, EDECIO - FM ; MADY, CHARLES - FM ; KALIL FILHO, JORGE ELIAS - FM ; IANNI, BARBARA MARIA - FM
- Unidade: FM
- DOI: 10.1016/j.molimm.2007.04.015
- Subjects: MIOCARDIOPATIA CHAGÁSICA; TRYPANOSOMA CRUZI (PATOGENICIDADE); POLIMORFISMO
- Language: Inglês
- Imprenta:
- Source:
- Título: Molecular Immunology
- ISSN: 0161-5890
- Volume/Número/Paginação/Ano: v. 45, n. 1, p. 283-288, 2008
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
RAMASAWMY, Rajendranath et al. Variants in the promoter region of IKBL/NFKBIL1 gene may mark susceptibility to the development of chronic Chagas' cardiomyopathy among Trypanosoma cruzi-infected individuals. Molecular Immunology, v. 45, n. 1, p. 283-288, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.molimm.2007.04.015. Acesso em: 13 maio 2025. -
APA
Ramasawmy, R., Fáe, K. C., Cunha Neto, E., Borba, S. C. P., Ianni, B. M., Mady, C., et al. (2008). Variants in the promoter region of IKBL/NFKBIL1 gene may mark susceptibility to the development of chronic Chagas' cardiomyopathy among Trypanosoma cruzi-infected individuals. Molecular Immunology, 45( 1), 283-288. doi:10.1016/j.molimm.2007.04.015 -
NLM
Ramasawmy R, Fáe KC, Cunha Neto E, Borba SCP, Ianni BM, Mady C, Goldberg AC, Kalil Filho JE. Variants in the promoter region of IKBL/NFKBIL1 gene may mark susceptibility to the development of chronic Chagas' cardiomyopathy among Trypanosoma cruzi-infected individuals [Internet]. Molecular Immunology. 2008 ; 45( 1): 283-288.[citado 2025 maio 13 ] Available from: https://doi.org/10.1016/j.molimm.2007.04.015 -
Vancouver
Ramasawmy R, Fáe KC, Cunha Neto E, Borba SCP, Ianni BM, Mady C, Goldberg AC, Kalil Filho JE. Variants in the promoter region of IKBL/NFKBIL1 gene may mark susceptibility to the development of chronic Chagas' cardiomyopathy among Trypanosoma cruzi-infected individuals [Internet]. Molecular Immunology. 2008 ; 45( 1): 283-288.[citado 2025 maio 13 ] Available from: https://doi.org/10.1016/j.molimm.2007.04.015 - Plasma cytokine profile in tropical endomyocardial fibrosis: Predominance of TNF-alfa, IL-4 and IL-10
- Heterozygosity for the S180L variant of Mal/TIRAP, a gene expressing an adaptor protein in the toll-like receptor pathway, is associated lower risk of developing chronic chagas cardiomyopathy
- Human long-term CD8+ T cell lines express lower levels of IL-2 receptor alpha and gamma chains than long-term CD4+ T cell lines from Chagas patients
- Locally produced IL-15 may underlie the predominance of CD8+ IL2Ralpha- T cells at heart lesions of human Chagas`disease cardiomyopathy
- TLR ligand stimulation of PBMC from Chagas'disease cardiomyopathy leads to increased TNF-alfa and IFN-y and decreased IL-10 production
- Increased plasma levels of TNF-'alfa' in Chagas disease and Chagas disease cardiomyopathy
- T-cell molecular mimicry in Chagas disease: identification and partial structural analysis of multiple cross-reactive epitopes between Trypanosoma cruzi B13 and cardiac myosin heavy chain
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- HIV CD4 based vaccine increases CD8 cell responses andconfers protection against challenge with recombinant vaccinia enconding HIV gag-pol proteins
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Informações sobre o DOI: 10.1016/j.molimm.2007.04.015 (Fonte: oaDOI API)
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