Wall shear stress and stretch differentially affect aorta remodeling in rats (2007)
- Authors:
- USP affiliated authors: RAMOS, SIMONE GUSMÃO - FMRP ; ROSSI, MARCOS ANTONIO - FMRP ; ELIAS JÚNIOR, JORGE - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- Assunto: ESTENOSE DA VALVA AÓRTICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Poster Sessions
- Conference titles: Congress of the European Atherosclerosis
-
ABNT
PRADO, C. M. et al. Wall shear stress and stretch differentially affect aorta remodeling in rats. 2007, Anais.. Helsinki: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2007. . Acesso em: 06 jun. 2025. -
APA
Prado, C. M., Ramos, S. G., Alves-Filho, J. C., Elias Júnior, J., Cunha, F. de Q., & Rossi, M. A. (2007). Wall shear stress and stretch differentially affect aorta remodeling in rats. In Poster Sessions. Helsinki: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Prado CM, Ramos SG, Alves-Filho JC, Elias Júnior J, Cunha F de Q, Rossi MA. Wall shear stress and stretch differentially affect aorta remodeling in rats. Poster Sessions. 2007 ;[citado 2025 jun. 06 ] -
Vancouver
Prado CM, Ramos SG, Alves-Filho JC, Elias Júnior J, Cunha F de Q, Rossi MA. Wall shear stress and stretch differentially affect aorta remodeling in rats. Poster Sessions. 2007 ;[citado 2025 jun. 06 ] - Aorta remodeling responses to hemodynamic stimuli induced by coarctation in rats
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- Hypertension, laminar flow and normal shear stress or normotension, turbulent flow and low shear stress cause distinct aorta remodeling
- Turbulent flow/low wall shear stress and stretch differentially affect aorta remodeling in rats
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- Color doppler ultrasound (CDUS) analysis of turbulent flow in three different animal models
- Chronic inhibition of nitric oxide synthase induces hypertension and cardiomyocyte mitochondrial and myocardial collagen remodelling in the absence of hypertrophy
- Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally-induced hypercholesterolemia in rats
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